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Article

Human papillomavirus in non-small-cell lung cancer: the impact of EGFR mutations and the response to erlotinib.

Archivos de bronconeumologia - 1 Feb 2013

Márquez-Medina Diego, Gasol-Cudós Ariadna, Taberner-Bonastre M Teresa, Samamé Pérez-Vargas Juan Carlos, Salud-Salvia Antonieta, Llombart-Cussac Antonio

Abstract excerpt

It has been suggested that human papillomavirus (HPV) could participate in the development of non-small-cell lung cancer (NSCLC). A higher HPV infection rate has been reported in the NSCLC samples from Asian non-smoker patients, with adenocarcinomas or responders to EGFR tyrosine kinase inhibitors (EGFR-TKI). We explored a potential relationship between EGFR mutation, response to EGFR-TKI and HPV infection in...

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Share to Adenocarcinoma, Bronchiolo-Alveolar
Share to Carcinoma, Squamous Cell

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Erlotinib Hydrochloride

Share to Genes, erbB-1

Share to Papillomavirus Infections

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Source

Identifiers

PMID: 22627040
DOI: 10.1016/j.arbres.2012.04.003

Genes

EGFR

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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