Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Analysis of CTLA4 gene variant in infertile Brazilian women with and without endometriosis.

International journal of immunogenetics - 1 Jun 2011

Lerner T G, Bianco B, Teles J S, Vilarino F L, Christofolini D M, Barbosa C P

Abstract excerpt

An autoimmune aetiology has been suggested for endometriosis mostly on the basis of an increased prevalence of autoimmune diseases in affected women. Cytotoxic T lymphocyte antigen (CTLA) 4 gene is recognized as a primary determinant for autoimmunity, since specific polymorphisms have been associated with predisposition to most autoimmune disorders. Thus, the objective of the study was to evaluate CTLA4...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Alleles
Share to Antigens, CD
Share to Brazil

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to CTLA-4 Antigen

Share to Endometriosis

Share to Gene Frequency

Share to Infertility, Female

Share to Polymorphism, Genetic

Source

Identifiers

PMID: 21599853
DOI: 10.1111/j.1744-313X.2011.01000.x

Genes

CTLA4

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Analysis of CTLA4 gene variant in infertile Brazilian women with and without endometriosis.PMID 21599853Read ↗

Variants of the CTLA4 gene that segregate with autoimmune diseases are not associated with endometriosis.Semantic proximity Read ↗

STAT4 single nucleotide gene polymorphisms and susceptibility to endometriosis-related infertility.Semantic proximity Read ↗

Association of FCRL3 -169T/C polymorphism with endometriosis and identification of a protective haplotype against the development of the disease in Brazilian population.Semantic proximity Read ↗

CTLA4 Gene Variants in Autoimmunity and Cancer: a Comparative ReviewSemantic proximity Read ↗

Epigenetic regulation and T-cell responses in endometriosis – something other than autoimmunitySemantic proximity Read ↗

Single nucleotide polymorphisms of Interleukin - 4, Interleukin-18, FCRL3 and sPLA2IIa genes and their association in pathogenesis of endometriosis.Semantic proximity Read ↗

Evaluation of CTLA-4 (+ 49A/G) polymorphism association with rheumatoid arthritis in Egyptian patients.Semantic proximity Read ↗

Cytotoxic T-lymphocyte-associated protein 4 gene polymorphism is related to rheumatoid arthritis in Egyptian population.Semantic proximity Read ↗

The effect of CTLA-4 A49G polymorphism on rheumatoid arthritis risk: a meta-analysis.Semantic proximity Read ↗

The possible role of genetic variants in autoimmune-related genes in the development of endometriosisSemantic proximity Read ↗

The role of CTLA-4 exon-1 49 A/G polymorphism and soluble CTLA-4 protein level in egyptian patients with Behçet's disease.Semantic proximity Read ↗

Cytotoxic T lymphocyte associated antigen-4 (CTLA-4) +49 A>G gene polymorphism in Egyptian cases with rheumatoid arthritis.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.