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Article

Strain‐specific vulnerability to alcohol exposure in utero via hippocampal parent‐of‐origin expression of deiodinase‐III

23 Mar 2011

Abstract excerpt

Prenatal exposure to alcohol is thought to be the most prevalent nongenetic cause of a wide range of neurodevelopmental deficits. Insufficient thyroid hormone levels are one mechanism that hampers development of the alcohol-exposed brain, and we hypothesized that altered dosage of the imprinted thyroid hormone-inactivating gene deiodinase-III (Dio3) is responsible. To follow parent-of-origin allelic expression of...

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PMID: 21429942
DOI: 10.1096/fj.10-179234

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