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Article

Destabilization of TIP60 by Human Papillomavirus E6 Results in Attenuation of TIP60-Dependent Transcriptional Regulation and Apoptotic Pathway

1 Jun 2010

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 20542002
DOI: 10.1016/j.molcel.2010.05.020

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Related research

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Destabilization of TIP60 by Human Papillomavirus E6 Results in Attenuation of TIP60-Dependent Transcriptional Regulation and Apoptotic PathwayPMID 20542002Read ↗

Degradation of p53 can be targeted by HPV E6 sequences distinct from those required for p53 binding and trans-activationSemantic proximity Read ↗

The E6 oncoprotein encoded by human papillomavirus types 16 and 18 promotes the degradation of p53Semantic proximity Read ↗

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Identification of cellular targets for the human papillomavirus E6 and E7 oncogenes by RNA interference and transcriptome analysesSemantic proximity Read ↗

Post-translational modifications of epigenetic modifier TIP60: their role in cellular functions and cancerSemantic proximity Read ↗

The multitalented TIP60 chromatin remodeling complex: wearing many hats in epigenetic regulation, cell division and diseasesSemantic proximity Read ↗

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Cellular Steady-State Levels of “High Risk” but Not “Low Risk” Human Papillomavirus (HPV) E6 Proteins Are Increased by Inhibition of Proteasome-Dependent Degradation Independent of Their p53- and E6AP-Binding CapabilitiesSemantic proximity Read ↗

Human papillomavirus E6 proteins bind p53 in vivo and abrogate p53‐mediated repression of transcription.Semantic proximity Read ↗

The stability of the human papillomavirus E6 oncoprotein is E6AP dependentSemantic proximity Read ↗

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