Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Lipopolysaccharide as shield and receptor for R-pyocin-mediated killing in Pseudomonas aeruginosa.

Journal of bacteriology - 1 Apr 2010

Köhler Thilo, Donner Viviane, van Delden Christian

Abstract excerpt

Pseudomonas aeruginosa produces three different types of bacteriocins: the soluble S-pyocins and the bacteriophage-like F- and R-pyocins. R-pyocins kill susceptible bacteria of the same or closely related species with high efficiency. Five different types of R-pyocins (R1- to R5-pyocins) have been described based on their killing spectra and tail fiber protein sequences. We analyzed the distribution of R-pyocin...

Topics

Anti-Bacterial Agents
Antibiosis
Humans
Lipopolysaccharides
Models, Molecular
Mutation
O Antigens
Pseudomonas Infections
Pseudomonas aeruginosa
Pyocins
Serotyping

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Open in PubMed Open full text

Identifiers

PMID: 20118263
DOI: 10.1128/JB.01459-09
PMCID: PMC2838038

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 29 Aug 2026. Prepared record publication-20118263-2026-08-29.1. Manifest publication-graph-cohort-2026-08-29.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 29 Aug 2026.

Community interpretation

Discuss the evidence

Ask a question, add context or challenge an interpretation. Community activity is separate from scientific evidence strength.

0

discussions

0

replies

Citations encouraged

Claims remain attributable.

No discussion yet

Be the first to add a careful, cited interpretation.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Lipopolysaccharide as shield and receptor for R-pyocin-mediated killing in Pseudomonas aeruginosa.PMID 20118263Read ↗

F-type Pyocins are Diverse Non-Contractile Phage Tail-Like Weapons for Killing <i>Pseudomonas aeruginosa</i>Semantic proximity Read ↗

Heterogenous susceptibility to R-pyocins in populations of <i>Pseudomonas aeruginosa</i> sourced from cystic fibrosis lungsSemantic proximity Read ↗

High prevalence of lipopolysaccharide mutants and R2-pyocin susceptible variants in Pseudomonas aeruginosa populations sourced from cystic fibrosis lung infectionsSemantic proximity Read ↗

R-type pyocin is required for competitive growth advantage between Pseudomonas aeruginosa strains.Semantic proximity Read ↗

Role of R5 Pyocin in the Predominance of High-Risk <i>Pseudomonas aeruginosa</i> IsolatesSemantic proximity Read ↗

High prevalence of lipopolysaccharide mutants and R2-Pyocin susceptible variants in <i>Pseudomonas aeruginosa</i> populations sourced from cystic fibrosis lung infectionsSemantic proximity Read ↗

R pyocin sensitivity of Pseudomonas aeruginosa clinical isolates from different disease types.Semantic proximity Read ↗

«Study of 32 new phage tail-like bacteriocins (pyocins) from a clinical collection of Pseudomonas aeruginosa and of their potential use as typing markers and antimicrobial agents»Semantic proximity Read ↗

Competition in biofilms between cystic fibrosis isolates of <i>Pseudomonas aeruginosa</i> is shaped by R-pyocinsSemantic proximity Read ↗

Chimeric Ligands of Pili and Lectin A Inhibit Tolerance, Persistence, and Virulence Factors of Pseudomonas aeruginosa over a Wide Range of Phenotypes.Semantic proximity Read ↗

Antibacterial Efficacy of R-Type Pyocins towards <i>Pseudomonas aeruginosa</i> in a Murine Peritonitis ModelSemantic proximity Read ↗

Intra-strain and inter-strain heterogeneity shape phage-host interactions and phenotypic adaptation in Pseudomonas aeruginosa.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.