Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

High prevalence of lipopolysaccharide mutants and R2-pyocin susceptible variants in Pseudomonas aeruginosa populations sourced from cystic fibrosis lung infections

25 Oct 2023

Abstract excerpt

ABSTRACT Chronic, highly antibiotic-resistant infections in cystic fibrosis (CF) lungs contribute to increasing morbidity and mortality. Pseudomonas aeruginosa , a common CF pathogen, exhibits resistance to multiple antibiotics, contributing to antimicrobial resistance (AMR). These bacterial populations display genetic and phenotypic diversity, but it is unclear how this diversity affects susceptibility to...

Source

Identifiers

PMID: 37877708
DOI: 10.1128/spectrum.01773-23

Provenance

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 30 Aug 2026.

Community interpretation

Discuss the evidence

Ask a question, add context or challenge an interpretation. Community activity is separate from scientific evidence strength.

0

discussions

0

replies

Citations encouraged

Claims remain attributable.

No discussion yet

Be the first to add a careful, cited interpretation.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

High prevalence of lipopolysaccharide mutants and R2-pyocin susceptible variants in Pseudomonas aeruginosa populations sourced from cystic fibrosis lung infectionsPMID 37877708Read ↗

High prevalence of lipopolysaccharide mutants and R2-Pyocin susceptible variants in <i>Pseudomonas aeruginosa</i> populations sourced from cystic fibrosis lung infectionsSemantic proximity Read ↗

Heterogenous susceptibility to R-pyocins in populations of <i>Pseudomonas aeruginosa</i> sourced from cystic fibrosis lungsSemantic proximity Read ↗

R pyocin sensitivity of Pseudomonas aeruginosa clinical isolates from different disease types.Semantic proximity Read ↗

Lipopolysaccharide as shield and receptor for R-pyocin-mediated killing in Pseudomonas aeruginosa.Semantic proximity Read ↗

Competition in biofilms between cystic fibrosis isolates of <i>Pseudomonas aeruginosa</i> is shaped by R-pyocinsSemantic proximity Read ↗

Role of R5 Pyocin in the Predominance of High-Risk <i>Pseudomonas aeruginosa</i> IsolatesSemantic proximity Read ↗

Competition in Biofilms between Cystic Fibrosis Isolates of Pseudomonas aeruginosa Is Shaped by R-PyocinsSemantic proximity Read ↗

Susceptibility to R-pyocins of Pseudomonas aeruginosa clinical isolates from cystic fibrosis patientsSemantic proximity Read ↗

F-type Pyocins are Diverse Non-Contractile Phage Tail-Like Weapons for Killing <i>Pseudomonas aeruginosa</i>Semantic proximity Read ↗

Global genomic diversity of <i>Pseudomonas aeruginosa</i> in bronchiectasisSemantic proximity Read ↗

Biofilm Formation of Pseudomonas aeruginosa in Cystic Fibrosis: Mechanisms of Persistence, Adaptation, and PathogenesisSemantic proximity Read ↗

Phenotypic characterization of an international Pseudomonas aeruginosa reference panel: strains of cystic fibrosis (CF) origin show less in vivo virulence than non-CF strains.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.