Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

LQTS mutation N1325S in cardiac sodium channel gene SCN5A causes cardiomyocyte apoptosis, cardiac fibrosis and contractile dysfunction in mice

19 Sept 2009

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cardiac electrophysiology and arrhythmias
Share to Ion channel regulation and function
Share to Ion Transport and Channel Regulation

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 19762097
DOI: 10.1016/j.ijcard.2009.08.047

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

LQTS mutation N1325S in cardiac sodium channel gene SCN5A causes cardiomyocyte apoptosis, cardiac fibrosis and contractile dysfunction in micePMID 19762097Read ↗

[The cardiac sodium channel gene SCN5A mutation and dilated cardiomyopathy].Semantic proximity Read ↗

Cardiac sodium channelopathy associated with SCN5A mutations: electrophysiological, molecular and genetic aspects.Semantic proximity Read ↗

Sodium channel mutations and arrhythmias.Semantic proximity Read ↗

SCN5A Mutation Associated With Dilated Cardiomyopathy, Conduction Disorder, and ArrhythmiaSemantic proximity Read ↗

The many faces of SCN5A pathogenic variants: from channelopathy to cardiomyopathy.Semantic proximity Read ↗

[Dilated cardiomyopathy caused by p.E446K mutation in SCN5A gene].Semantic proximity Read ↗

A transgenic zebrafish model of a human cardiac sodium channel mutation exhibits bradycardia, conduction-system abnormalities and early death.Semantic proximity Read ↗

Novel idiopathic DCM-related SCN5A variants localised in DI-S4 predispose electrical disorders by reducing peak sodium current density.Semantic proximity Read ↗

A leaky voltage sensor domain of cardiac sodium channels causes arrhythmias associated with dilated cardiomyopathy.Semantic proximity Read ↗

Dilated cardiomyopathy is associated with reduced expression of the cardiac sodium channel Scn5a.Semantic proximity Read ↗

Cardiac conduction defects associate with mutations in SCN5A.Semantic proximity Read ↗

SCN5A channelopathy: arrhythmia, cardiomyopathy, epilepsy and beyond.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.