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Article

Involvement of CHOP, an ER-stress apoptotic mediator, in both human sporadic ALS and ALS model mice

5 Sept 2009

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Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 19733664
DOI: 10.1016/j.nbd.2009.08.013

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Related research

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Involvement of CHOP, an ER-stress apoptotic mediator, in both human sporadic ALS and ALS model micePMID 19733664Read ↗

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Transcription Factor C/EBP Homologous Protein in Health and DiseasesSemantic proximity Read ↗

CHOP Potentially Co-Operates with FOXO3a in Neuronal Cells to Regulate PUMA and BIM Expression in Response to ER StressSemantic proximity Read ↗

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ER stress and neurodegenerative diseasesSemantic proximity Read ↗

ER Stress and UPR in Familial Amyotrophic Lateral SclerosisSemantic proximity Read ↗

Deletion of the BH3-only protein <i>puma</i> protects motoneurons from ER stress-induced apoptosis and delays motoneuron loss in ALS miceSemantic proximity Read ↗

Stress signaling from the endoplasmic reticulum: A central player in the pathogenesis of amyotrophic lateral sclerosisSemantic proximity Read ↗

DJ-1 modulates the unfolded protein response and cell death via upregulation of ATF4 following ER stressSemantic proximity Read ↗

The unfolded protein response modulates disease severity in Pelizaeus-Merzbacher disease.Semantic proximity Read ↗

DR5 and caspase-8 are dispensable in ER stress-induced apoptosisSemantic proximity Read ↗

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