Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Low N-acetyltransferase 2 activity in isoniazid-associated acute hepatitis requiring liver transplantation.

Transplant international : official journal of the European Society for Organ Transplantation - 1 Feb 2010

Cramer Jakob P, Lohse Ansgar W, Burchard Gerd D, Fischer Lutz, Nashan Björn, Zimmermann Mathias, Marx Andreas, Kluge Stefan

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Antitubercular Agents
Share to Arylamine N-Acetyltransferase
Share to Chemical and Drug Induced Liver Injury

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Genes, MDR

Share to Heterozygote

Share to Isoniazid

Share to Liver Transplantation

Share to Risk Factors

Source

Identifiers

PMID: 19686464
DOI: 10.1111/j.1432-2277.2009.00921.x

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 19 Oct 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Low N-acetyltransferase 2 activity in isoniazid-associated acute hepatitis requiring liver transplantation.PMID 19686464Read ↗

N-acetyl transferase 2 and cytochrome P450 2E1 genes and isoniazid-induced hepatotoxicity in Brazilian patients.Semantic proximity Read ↗

Genetic polymorphisms of drug-metabolizing enzymes and the susceptibility to antituberculosis drug-induced liver injury.Semantic proximity Read ↗

Slow N-acetyltransferase 2 genotype affects the incidence of isoniazid and rifampicin-induced hepatotoxicity.Semantic proximity Read ↗

N-acetyltransferase 2 (NAT2) genotype as a risk factor for development of drug-induced liver injury relating to antituberculosis drug treatment in a mixed-ethnicity patient group.Semantic proximity Read ↗

Polymorphism of the N-acetyltransferase 2 gene as a susceptibility risk factor for antituberculosis drug-induced hepatitis.Semantic proximity Read ↗

[Incidence of hepatic changes in relation to the isoniazid acetylator phenotype].Semantic proximity Read ↗

The incidence of liver injury in Uyghur patients treated for TB in Xinjiang Uyghur autonomous region, China, and its association with hepatic enzyme polymorphisms nat2, cyp2e1, gstm1 and gstt1.Semantic proximity Read ↗

Isoniazid acetylation rate and development of antinuclear antibodies upon isoniazid treatment.Semantic proximity Read ↗

Population pharmacokinetic analysis of isoniazid, acetylisoniazid, and isonicotinic acid in healthy volunteers.Semantic proximity Read ↗

Association of isoniazid-metabolizing enzyme genotypes and isoniazid-induced hepatotoxicity in tuberculosis patients.Semantic proximity Read ↗

N-Acetyltransferase2 genotype correlated with isoniazid acetylation in Japanese tuberculous patients.Semantic proximity Read ↗

Dose-escalation study of isoniazid in healthy volunteers with the rapid acetylator genotype of arylamine N-acetyltransferase 2.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.