Case-control association study of 65 candidate genes revealed a possible association of a SNP of HTR5A to be a factor susceptible to bipolar disease in Bulgarian population.

doi:10.1016/j.jad.2008.12.021

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Community

Back to search

Article

Case-control association study of 65 candidate genes revealed a possible association of a SNP of HTR5A to be a factor susceptible to bipolar disease in Bulgarian population.

Journal of affective disorders - 1 Sept 2009

Yosifova Adelina, Mushiroda Taisei, Stoianov Drozdstoi, Vazharova Radoslava, Dimova Ivanka, Karachanak Sena, Zaharieva Irina, Milanova Vihra, Madjirova Nadejda, Gerdjikov Ivan, Tolev Todor, Velkova Stoyanka, Kirov George, Owen Michael J, O'Donovan Michael C, Toncheva Draga, Nakamura Yusuke

Abstract excerpt

BACKGROUND: Bipolar affective disorder (BAD) is a psychiatric illness characterized by episodes of mania and depression. Although the etiology is not clear, epidemiological studies suggest it is a result of an interaction of genetic and environmental factors. Despite of enormous efforts and abundant studies conducted, none has yet been identified definitively a gene susceptible to bipolar disorder. METHODS:...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Bipolar Disorder

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Bulgaria

Share to Case-Control Studies

Share to Genetic Association Studies

Share to Genetic Predisposition to Disease

Share to Polymorphism, Single Nucleotide

Share to Receptors, Serotonin

Share to Risk Factors

Source

Open in PubMed

Identifiers

PMID: 19328558
DOI: 10.1016/j.jad.2008.12.021

Genes

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Case-control association study of 65 candidate genes revealed a possible association of a SNP of HTR5A to be a factor susceptible to bipolar disease in Bulgarian population.PMID 19328558Read ↗

Genome-wide association study on bipolar disorder in the Bulgarian population.Semantic proximity Read ↗

Association between the GABA(A) receptor alpha5 subunit gene locus (GABRA5) and bipolar affective disorder.Semantic proximity Read ↗

The genetics of bipolar disorder.Semantic proximity Read ↗

A susceptibility locus for bipolar affective disorder on chromosome 4q35.Semantic proximity Read ↗

[Genetics of bipolar affective disorders. Current status of research for identification of susceptibility genes].Semantic proximity Read ↗

Case-control association study of GRIA1, GRIA2 and GRIA4 polymorphisms in bipolar disorder.Semantic proximity Read ↗

Genetics of bipolar disorder.Semantic proximity Read ↗

Identification of new putative susceptibility genes for several psychiatric disorders by association analysis of regulatory and non-synonymous SNPs of 306 genes involved in neurotransmission and neurodevelopment.Semantic proximity Read ↗

Association studies of candidate genes in bipolar disorders.Semantic proximity Read ↗

Association between GABA-A receptor alpha 5 subunit gene locus and schizophrenia of a later age of onset.Semantic proximity Read ↗

Whole-genome-wide association study in the Bulgarian population reveals HHAT as schizophrenia susceptibility gene.Semantic proximity Read ↗

GABA-A receptor beta3 and alpha5 subunit gene cluster on chromosome 15q11-q13 and bipolar disorder: a genetic association study.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.