Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Mathematical analysis of a HIV model with frequency dependence and viral diversity.

Mathematical biosciences and engineering : MBE - 1 Jul 2008

Iwami Shingo, Nakaoka Shinji, Takeuchi Yasuhiro

Abstract excerpt

We consider the effect of viral diversity on the human immune system with the frequency dependent proliferation rate of CTLs and the elimination rate of infected cells by CTLs. The model has very complex mathematical structures such as limit cycle, quasi-periodic attractors, chaotic attractors, a...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Acquired Immunodeficiency Syndrome
Share to Algorithms
Share to CD4-Positive T-Lymphocytes

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to HIV Infections

Share to Immune System

Share to Mathematics

Share to Models, Statistical

Share to Models, Theoretical

Source

Identifiers

PMID: 18616352
DOI: 10.3934/mbe.2008.5.457

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026. Prepared record publication-18616352-2026-09-04.2. Manifest publication-graph-cohort-2026-09-04.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 4 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Mathematical analysis of a HIV model with frequency dependence and viral diversity.PMID 18616352Read ↗

The effects of CTL immune response on HIV infection model with potent therapy, latently infected cells and cell-to-cell viral transmissionSemantic proximity Read ↗

Virus dynamic behavior of a stochastic HIV/AIDS infection model including two kinds of target cell infections and CTL immune responsesSemantic proximity Read ↗

A mathematical model for HIV dynamics with multiple infections: implications for immune escape.Semantic proximity Read ↗

Global Analysis for an HIV Infection Model with CTL Immune Response and Infected Cells in Eclipse PhaseSemantic proximity Read ↗

Mathematical modeling of hepatitis B virus infection incorporating immune responsesSemantic proximity Read ↗

Global properties of a cell mediated immunity in HIV infection model with two classes of target cells and distributed delaysSemantic proximity Read ↗

Mathematical analysis of a class of HIV infection models of CD4+ T-cells with combined antiretroviral therapySemantic proximity Read ↗

Modeling and analysis of a periodic delays spatialdiffusion HIV model with three-stage infection processSemantic proximity Read ↗

Bifurcation analysis of a multidelayed HIV model in presence of immune response and understanding of in‐host viral dynamicsSemantic proximity Read ↗

Qualitative analysis of a novel HIV infection model of macrophage cells in Caputo fractional environmentSemantic proximity Read ↗

Population dynamics of immune responses to persistent viruses.Semantic proximity Read ↗

A modeling approach to the impact of HIV mutations on the immune system.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.