Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Bloom's Syndrome Helicase and Mus81 are Required to Induce Transient Double-strand DNA Breaks in Response to DNA Replication Stress

14 Nov 2007

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Acute Lymphoblastic Leukemia research
Share to Carcinogens and Genotoxicity Assessment
Share to DNA Repair Mechanisms

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 18054789
DOI: 10.1016/j.jmb.2007.11.006

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Bloom's Syndrome Helicase and Mus81 are Required to Induce Transient Double-strand DNA Breaks in Response to DNA Replication StressPMID 18054789Read ↗

Ubiquitin‐dependent recruitment of the Bloom Syndrome helicase upon replication stress is required to suppress homologous recombinationSemantic proximity Read ↗

RADX interacts with single‐stranded DNA to promote replication fork stabilitySemantic proximity Read ↗

FBH1 co-operates with MUS81 in inducing DNA double-strand breaks and cell death following replication stress.Semantic proximity Read ↗

RAD51AP1 -deficiency in vertebrate cells impairs DNA replicationSemantic proximity Read ↗

Causes and consequences of DNA single-strand breaksSemantic proximity Read ↗

DNA single-strand break repairSemantic proximity Read ↗

MCM9 Is Required for Mammalian DNA Mismatch RepairSemantic proximity Read ↗

DNA replication stress: from molecular mechanisms to human diseaseSemantic proximity Read ↗

DNA replication stress: Causes, resolution and diseaseSemantic proximity Read ↗

DNA double-strand break signaling and human disordersSemantic proximity Read ↗

RecQ helicases and PARP1 team up in maintaining genome integritySemantic proximity Read ↗

ATR and ATM differently regulate WRN to prevent DSBs at stalled replication forks and promote replication fork recoverySemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.