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Article

ACE gene deletion/deletion polymorphism may be a protective factor for respiratory distress in preterm infants.

The Turkish journal of pediatrics - 1 Jan 2000

Sivasli Ercan, Yurdakök Murat, Babaoğlu Elif, Karabulut Halil, Yiğit Sule, Babaoğlu Melih, Tekinalp Gülsevin, Tükün Ajlan

Abstract excerpt

The objective in this study was to evaluate the angiotensin converting enzyme (ACE) insertion/deletion (I/D) gene polymorphism in premature infants with and without respiratory distress within the first 24 hours of life. Totally, 87 premature babies who were followed up in the neonatal unit were included in the study. Of these babies, 41 had respiratory distress, and constituted the patient group. The remaining...

Read the complete abstract on PubMed

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Share to Gene Deletion
Share to Infant, Newborn

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Infant, Premature

Share to Intensive Care Units, Neonatal

Share to Peptidyl-Dipeptidase A

Share to Polymorphism, Genetic

Share to Respiratory Distress Syndrome, Newborn

Source

Identifiers

PMID: 17479647

Genes

ACE

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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