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Article

The relationship between c-FLIP expression and human papillomavirus E2 gene disruption in cervical carcinogenesis

13 Apr 2007

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Share to Cell death mechanisms and regulation
Share to Cervical Cancer and HPV Research
Share to Toxin Mechanisms and Immunotoxins

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 17433827
DOI: 10.1016/j.ygyno.2007.01.051

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Related research

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The relationship between c-FLIP expression and human papillomavirus E2 gene disruption in cervical carcinogenesisPMID 17433827Read ↗

Triggering of death receptor apoptotic signaling by human papillomavirus 16 E2 protein in cervical cancer cell lines is mediated by interaction with c-FLIPSemantic proximity Read ↗

The antiapoptotic protein, FLIP, is regulated by heterogeneous nuclear ribonucleoprotein K and correlates with poor overall survival of nasopharyngeal carcinoma patientsSemantic proximity Read ↗

Loss of expression, and mutations of Smad 2 and Smad 4 in human cervical cancerSemantic proximity Read ↗

c-FLIP expression in bladder urothelial carcinomas: its role in resistance to Fas-mediated apoptosis and clinicopathologic correlations.Semantic proximity Read ↗

FLIP as an Anti-Cancer Therapeutic TargetSemantic proximity Read ↗

Aberrant FHIT transcripts in squamous cell carcinoma of the uterine cervix.Semantic proximity Read ↗

Epigenetics and cervical cancer: from pathogenesis to therapySemantic proximity Read ↗

Overview of genetic and epigenetic regulation of human papillomavirus and apoptosis in cervical cancerSemantic proximity Read ↗

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