Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Variation in expression of Biomphalaria glabrata SOD1: a potential controlling factor in susceptibility/resistance to Schistosoma mansoni.

Developmental and comparative immunology - 1 Jan 2007

Bender Randall C, Goodall Cheri P, Blouin Michael S, Bayne Christopher J

Abstract excerpt

The snail Biomphalaria glabrata kills the blood fluke Schistosoma mansoni by a mechanism involving production of hydrogen peroxide, the enzymatic product of cytosolic Cu/Zn superoxide dismutase (SOD1). This enzyme exhibits higher activity in blood cells (hemocytes) from a predominantly resistant strain of B. glabrata than in hemocytes from a susceptible strain. Additionally, B. glabrata SOD1 polymorphisms have...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Biomphalaria
Share to Disease Susceptibility

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Gene Expression Regulation, Enzymologic

Share to RNA, Messenger

Share to Schistosoma mansoni

Share to Schistosomiasis mansoni

Share to Superoxide Dismutase

Share to Superoxide Dismutase-1

Source

Identifiers

PMID: 17292470
DOI: 10.1016/j.dci.2006.12.005

Genes

SOD1

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Variation in expression of Biomphalaria glabrata SOD1: a potential controlling factor in susceptibility/resistance to Schistosoma mansoni.PMID 17292470Read ↗

Biomphalaria glabrata cytosolic copper/zinc superoxide dismutase (SOD1) gene: association of SOD1 alleles with resistance/susceptibility to Schistosoma mansoni.Semantic proximity Read ↗

Allelic variation partially regulates galactose-dependent hydrogen peroxide release from circulating hemocytes of the snail Biomphalaria glabrata.Semantic proximity Read ↗

Molecular studies of Biomphalaria glabrata, an intermediate host of Schistosoma mansoni.Semantic proximity Read ↗

The identification of markers segregating with resistance to Schistosoma mansoni infection in the snail Biomphalaria glabrata.Semantic proximity Read ↗

Effects of Cu/Zn superoxide dismutase (sod1) genotype and genetic background on growth, reproduction and defense in Biomphalaria glabrata.Semantic proximity Read ↗

Biomphalaria glabrata immunity: Post-genome advancesSemantic proximity Read ↗

Parasite-susceptibility phenotypes of F1 Biomphalaria glabrataprogeny derived from interbreeding Schistosoma mansoni-resistant and -susceptible snails.Semantic proximity Read ↗

Genetic markers between Biomphalaria glabrata snails susceptible and resistant to Schistosoma mansoni infection.Semantic proximity Read ↗

Clearance of schistosome parasites by resistant genotypes at a single genomic region in Biomphalaria glabrata snails involves cellular components of the hemolymph.Semantic proximity Read ↗

Coordination of humoral immune factors dictates compatibility between <i>Schistosoma mansoni</i> and <i>Biomphalaria glabrata</i>Semantic proximity Read ↗

Allelic Variation in a Single Genomic Region Alters the Microbiome of the Snail Biomphalaria glabrata.Semantic proximity Read ↗

Combining a transcriptomic approach and a targeted metabolomics approach for deciphering the molecular bases of compatibility phenotype in the snail Biomphalaria glabrata toward Schistosoma mansoni.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.