Immortalization by human papillomavirus type 16 alters retinoid regulation of human ectocervical epithelial cell differentiation.

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Article

Immortalization by human papillomavirus type 16 alters retinoid regulation of human ectocervical epithelial cell differentiation.

Cancer research - 1 Aug 1991

Agarwal C, Rorke E A, Irwin J C, Eckert R L

Abstract excerpt

Human cervical cells are a primary site of papillomavirus infection and 90% of all cervical tumors are positive for human papillomavirus (HPV) DNA. Over one-half million cases of HPV-associated cervical, vulvar, and penile cancers are reported per year. Yet, in spite of the magnitude of this prob...

Topics

Cell Differentiation
Cell Transformation, Viral
Cells, Cultured
Cervix Uteri
Epithelial Cells
Epithelium
Female
Humans
Keratins
Papillomaviridae
Phenotype
RNA, Messenger
Retinoids
Transcription, Genetic
Transfection
Tretinoin
Uterine Cervical Neoplasms
Virus Replication

Source

Open in PubMed

Identifiers

PMID: 1713124

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 30 Aug 2026. Prepared record publication-1713124-2026-08-30.1. Manifest publication-graph-cohort-2026-08-30.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 30 Aug 2026.

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