Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Interrelation of hyperhomocysteinemia and inherited risk factors for venous thromboembolism. Results from the E.D.I.TH. study: a hospital-based case-control study.

Thrombosis research - 1 Jan 2007

Oger Emmanuel, Lacut Karine, Le Gal Grégoire, Couturaud Francis, Abalain Jean-Hervé, Mercier Bernard, Mottier Dominique

Abstract excerpt

BACKGROUND: Moderate hyperhomocysteinemia and factor V Leiden mutation are among the most prevalent risk factors for venous thromboembolism (VTE). The hypothesis of an interaction between those risks has been raised and conflicting results were reported. METHODS: We designed a hospital-based case-control study to test the interaction between Factor V Leiden and fasting serum total homocysteine (tHcy). We have...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Factor V
Share to France

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Hyperhomocysteinemia

Share to Point Mutation

Share to Pregnancy

Share to Prothrombin

Share to Thromboembolism

Share to Venous Thrombosis

Source

Identifiers

PMID: 17126889
DOI: 10.1016/j.thromres.2006.10.002

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Interrelation of hyperhomocysteinemia and inherited risk factors for venous thromboembolism. Results from the E.D.I.TH. study: a hospital-based case-control study.PMID 17126889Read ↗

Interaction between hyperhomocysteinemia and inherited thrombophilic factors in venous thromboembolism.Semantic proximity Read ↗

No interaction between factor V Leiden and hyperhomocysteinemia or MTHFR 677TT genotype in venous thrombosis. Results of a meta-analysis of published studies and a large case-only study.Semantic proximity Read ↗

Importance of hyperhomocysteinemia as a risk factor for venous thromboembolism in a Taiwanese population. A case-control study.Semantic proximity Read ↗

Hyperhomocysteinemia and low B vitamin levels are independently associated with venous thromboembolism: results from the EDITH study: a hospital-based case-control study.Semantic proximity Read ↗

Hyperhomocysteinemia and venous thromboembolism: a risk factor more prevalent in the elderly and in idiopathic cases.Semantic proximity Read ↗

Interaction Between Hyperhomocysteinemia, Mutated Methylenetetrahydrofolatereductase (MTHFR) and Inherited Thrombophilic Factors in Recurrent Venous ThrombosisSemantic proximity Read ↗

Hyperhomocysteinemia: no longer a consideration in the management of venous thromboembolism.Semantic proximity Read ↗

The risk of venous thromboembolism associated with the factor V Leiden mutation and low B-vitamin status.Semantic proximity Read ↗

Intermediate and severe hyperhomocysteinemia with thrombosis: a study of genetic determinants.Semantic proximity Read ↗

Hyperhomocysteinemia and other inherited prothrombotic conditions in young adults with a history of ischemic stroke.Semantic proximity Read ↗

[Effect of hyperhomocysteinemia and methylenetetrahydrofolate reductase 677C --> T mutation in venous thromboembolism risk of young adults].Semantic proximity Read ↗

Hyperhomocysteinemia increases the risk of venous thrombosis independent of the C677T mutation of the methylenetetrahydrofolate reductase gene in selected Brazilian patients.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.