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Article

Complementation assays adapted for DNA repair-deficient keratinocytes.

Methods in molecular biology (Clifton, N.J.) - 1 Jan 2006

Fréchet Mathilde, Bergoglio Valérie, Chevallier-Lagente Odile, Sarasin Alain, Magnaldo Thierry

Abstract excerpt

Genetic alterations affecting nucleotide excision repair, the most versatile DNA-repair mechanism responsible for removal of bulky DNA adducts including ultraviolet (UV) light-induced DNA lesions, may result in the rare, recessively inherited autosomal syndromes xeroderma pigmentosum (XP), Cockayne syndrome (CS), or trichothiodystrophy (TTD). Classical approaches such as somatic cell fusions or microinjection...

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Identifiers

PMID: 16673870
DOI: 10.1385/1-59259-973-7:009

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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Complementation assays adapted for DNA repair-deficient keratinocytes.PMID 16673870Read ↗

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