The mutation in the ATP-binding region of JAK1, identified in human uterine leiomyosarcomas, results in defective interferon-γ inducibility of TAP1 and LMP2

doi:10.1038/sj.onc.1209434

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Community

Back to search

Article

The mutation in the ATP-binding region of JAK1, identified in human uterine leiomyosarcomas, results in defective interferon-γ inducibility of TAP1 and LMP2

13 Feb 2006

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Immune Cell Function and Interaction
Share to Immunotherapy and Immune Responses
Share to Lymphoma Diagnosis and Treatment

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Open in PubMed

Identifiers

PMID: 16474838
DOI: 10.1038/sj.onc.1209434

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 8 closest

The mutation in the ATP-binding region of JAK1, identified in human uterine leiomyosarcomas, results in defective interferon-γ inducibility of TAP1 and LMP2PMID 16474838Read ↗

Molecular Pathology and Novel Clinical Therapy for Uterine Leiomyosarcoma.Semantic proximity Read ↗

JAK1 truncating mutations in gynecologic cancer define new role of cancer-associated protein tyrosine kinase aberrations.Semantic proximity Read ↗

Fusion of CHOP to a novel RNA-binding protein in human myxoid liposarcomaSemantic proximity Read ↗

Potential role of LMP2 as tumor-suppressor defines new targets for uterine leiomyosarcoma therapy.Semantic proximity Read ↗

ATP binding by monarch-1/NLRP12 is critical for its inhibitory function.Semantic proximity Read ↗

Mitogen-activated protein kinases interacting kinases are autoinhibited by a reprogrammed activation segment.Semantic proximity Read ↗

Crystal structures of proto-oncogene kinase Pim1: a target of aberrant somatic hypermutations in diffuse large cell lymphoma.Semantic proximity Read ↗

Gene symbol: MLH1.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.