Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Regulatory role for Krüppel-like zinc-finger protein Gli-similar 1 (Glis1) in PMA-treated and psoriatic epidermis.

The Journal of investigative dermatology - 1 Jan 2006

Nakanishi Gen, Kim Yong-Sik, Nakajima Takeshi, Jetten Anton M

Abstract excerpt

In this study, we analyze the expression and potential function of the Krüppel-like zinc-finger protein Gli-similar protein 1 (Glis1) in normal and inflammatory skin and in the differentiation of epidermal keratinocytes. Glis1 mRNA is not expressed in normal human epidermis, but is significantly induced in psoriatic epidermis and in mouse skin upon treatment with the tumor promoter phorbol-12-myristate-13-acetate...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cell Nucleus
Share to Cytoplasm
Share to Epidermal Cells

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Epidermis

Share to Interferon-gamma

Share to Keratinocytes

Share to Psoriasis

Share to Tetradecanoylphorbol Acetate

Source

Open in PubMed Open full text

Identifiers

PMID: 16417217
DOI: 10.1038/sj.jid.5700018
PMCID: PMC1435652

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Regulatory role for Krüppel-like zinc-finger protein Gli-similar 1 (Glis1) in PMA-treated and psoriatic epidermis.PMID 16417217Read ↗

Epigenetic Mechanisms of Epidermal DifferentiationSemantic proximity Read ↗

The Potential Role of Krüppel-Like Zinc-Finger Protein Glis3 in Genetic Diseases and Cancers.Semantic proximity Read ↗

Grainyhead-like 2 (GRHL2) inhibits keratinocyte differentiation through epigenetic mechanismSemantic proximity Read ↗

miR-155 Contributes to Normal Keratinocyte Differentiation and Is Upregulated in the Epidermis of Psoriatic Skin LesionsSemantic proximity Read ↗

HDAC1/2 Control Proliferation and Survival in Adult Epidermis and Pre‒Basal Cell Carcinoma through p16 and p53Semantic proximity Read ↗

The Krüppel-Like Protein Gli-Similar 3 (Glis3) Functions as a Key Regulator of Insulin TranscriptionSemantic proximity Read ↗

Activation of the transcription factor Gli1 and the Sonic hedgehog signalling pathway in skin tumours.Semantic proximity Read ↗

The Bmi-1 helix-turn and ring finger domains are required for Bmi-1 antagonism of (-) epigallocatechin-3-gallate suppression of skin cancer cell survival.Semantic proximity Read ↗

Notch Signaling May Be Involved in the Abnormal Differentiation of Epidermal Keratinocytes in PsoriasisSemantic proximity Read ↗

The helix-loop-helix transcription factor Id1 is highly expressed in psoriatic involved skin.Semantic proximity Read ↗

The Krüppel-like zinc finger protein Glis3 directly and indirectly activates insulin gene transcriptionSemantic proximity Read ↗

The zinc-finger transcription factor GLI2 antagonizes contact inhibition and differentiation of human epidermal cellsSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.