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Article

A p53-dominant transcriptional response to cisplatin in testicular germ cell tumor-derived human embyronal carcinoma

23 May 2005

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Share to Epigenetics and DNA Methylation
Share to Histone Deacetylase Inhibitors Research
Share to Testicular diseases and treatments

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 15940259
DOI: 10.1038/sj.onc.1208755

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A p53-dominant transcriptional response to cisplatin in testicular germ cell tumor-derived human embyronal carcinomaPMID 15940259Read ↗

Molecular Mechanisms of Cisplatin Chemoresistance and Its Circumventing in Testicular Germ Cell TumorsSemantic proximity Read ↗

Towards an understanding of the biological basis of response to cisplatin-based chemotherapy in germ-cell tumorsSemantic proximity Read ↗

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The Role of TP53 in Cisplatin Resistance in Mediastinal and Testicular Germ Cell Tumors.Semantic proximity Read ↗

Cisplatin-induced apoptosis in human malignant testicular germ cell lines depends on MEK/ERK activationSemantic proximity Read ↗

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p53 Hypersensitivity Is the Predominant Mechanism of the Unique Responsiveness of Testicular Germ Cell Tumor (TGCT) Cells to CisplatinSemantic proximity Read ↗

Pro- and anti-apoptotic effects of p53 in cisplatin-treated human testicular cancer are cell context-dependentSemantic proximity Read ↗

Defective repair of cisplatin-induced DNA damage caused by reduced XPA protein in testicular germ cell tumoursSemantic proximity Read ↗

Influence of vitamin D on cisplatin sensitivity in testicular germ cell cancer-derived cell lines and in a NTera2 xenograft modelSemantic proximity Read ↗

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