Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Association of mannose binding lectin (MBL) gene polymorphism and serum MBL concentration with characteristics and progression of systemic lupus erythematosus.

Annals of the rheumatic diseases - 1 Feb 2005

Takahashi R, Tsutsumi A, Ohtani K, Muraki Y, Goto D, Matsumoto I, Wakamiya N, Sumida T

Abstract excerpt

OBJECTIVE: To determine whether occurrence, characteristics, and progression of systemic lupus erythematosus (SLE) are associated with polymorphism of the mannose binding lectin (MBL) gene and with serum MBL concentration. METHODS: Codon 54 MBL gene polymorphism of 147 patients with SLE and 160 healthy controls was determined by polymerase chain reaction-restriction fragment length polymorphism. Serum...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Adolescent
Share to Disease Progression

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Immunosuppressive Agents

Share to Lupus Erythematosus, Systemic

Share to Mannose-Binding Lectin

Share to Methylprednisolone

Share to Opportunistic Infections

Share to Polymorphism, Genetic

Source

Open in PubMed Open full text

Identifiers

PMID: 15647440
DOI: 10.1136/ard.2003.020172
PMCID: PMC1755352

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Association of mannose binding lectin (MBL) gene polymorphism and serum MBL concentration with characteristics and progression of systemic lupus erythematosus.PMID 15647440Read ↗

Mannose-binding lectin polymorphisms and susceptibility to infection in systemic lupus erythematosus.Semantic proximity Read ↗

The mannose-binding lectin gene polymorphisms and systemic lupus erythematosus: two case-control studies and a meta-analysis.Semantic proximity Read ↗

Association of mannose-binding lectin gene variation with disease severity and infections in a population-based cohort of systemic lupus erythematosus patients.Semantic proximity Read ↗

Mannose binding lectin (MBL) 2 gene polymorphism & its association with clinical manifestations in systemic lupus erythematosus (SLE) patients from western India.Semantic proximity Read ↗

Mannose-binding lectin gene: polymorphisms in Japanese patients with systemic lupus erythematosus, rheumatoid arthritis and Sjögren's syndrome.Semantic proximity Read ↗

[The role of mannose binding lectin in the pathogenesis of systemic lupus erythematosus].Semantic proximity Read ↗

Mannose-Binding Lectin Serum Level and Gene Polymorphism in Systemic Lupus Erythematosus Egyptian Patients.Semantic proximity Read ↗

Mannose-binding lectin (MBL) codon 54 (rs1800450) polymorphism predisposes towards medium vessel vasculitis in patients with systemic lupus erythematosus.Semantic proximity Read ↗

Mannose-binding lectin expression genotype in pediatric-onset systemic lupus erythematosus: associations with susceptibility to renal disease and protection against infections.Semantic proximity Read ↗

Mannose-binding lectin is a disease-modifying factor in North American patients with systemic lupus erythematosus.Semantic proximity Read ↗

Association of systemic lupus erythematosus with promoter polymorphisms of the mannose-binding lectin gene.Semantic proximity Read ↗

The association between the mannose-binding lectin codon 54 polymorphism and systemic lupus erythematosus: a meta-analysis update.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.