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Article

Histone deacetylase inhibitors induce a senescence-like state in human cells by a p16-dependent mechanism that is independent of a mitotic clock

3 Mar 2004

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Share to Epigenetics and DNA Methylation
Share to Histone Deacetylase Inhibitors Research
Share to Telomeres, Telomerase, and Senescence

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 15093749
DOI: 10.1016/j.yexcr.2004.01.017

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Histone deacetylase inhibitors induce a senescence-like state in human cells by a p16-dependent mechanism that is independent of a mitotic clockPMID 15093749Read ↗

Dysregulation of histone deacetylases in carcinogenesis and tumor progression: a possible link to apoptosis and autophagySemantic proximity Read ↗

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DNA Topoisomerase Inhibitors Induce Reversible Senescence in Normal Human FibroblastsSemantic proximity Read ↗

Telomere Shortening Triggers Senescence of Human Cells through a Pathway Involving ATM, p53, and p21CIP1, but Not p16INK4aSemantic proximity Read ↗

Human Fibroblast Commitment to a Senescence-Like State in Response to Histone Deacetylase Inhibitors Is Cell Cycle DependentSemantic proximity Read ↗

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