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Article

Evasion of Cellular Antiviral Responses by Human Cytomegalovirus TRS1 and IRS1

11 Dec 2003

Abstract excerpt

During infection with human cytomegalovirus (HCMV), cellular protein synthesis continues even as viral proteins are being synthesized in abundance. Thus, HCMV may have a mechanism for counteracting host cell antiviral pathways that act by shutting off translation. Consistent with this view, HCMV infection of human fibroblasts rescues the replication of a vaccinia virus mutant lacking the double-stranded...

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Identifiers

PMID: 14671101
DOI: 10.1128/jvi.78.1.197-205.2004

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

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