Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Localization of Familial Benign Hypercalcemia, Oklahoma Variant (FBHOk), to Chromosome 19q13

1 Jan 1999

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Asthma and respiratory diseases
Share to Biochemical Analysis and Sensing Techniques
Share to Olfactory and Sensory Function Studies

Source

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Identifiers

PMID: 9915958
DOI: 10.1086/302202

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 8 closest

Localization of Familial Benign Hypercalcemia, Oklahoma Variant (FBHOk), to Chromosome 19q13PMID 9915958Read ↗

Comparison of human chromosome 19q13 and syntenic region on mouse chromosome 7 reveals absence, in man, of 11.6 Mb containing four mouse calcium-sensing receptor-related sequences: relevance to familial benign hypocalciuric hypercalcaemia type 3.Semantic proximity Read ↗

Identification of a Second Kindred with Familial Hypocalciuric Hypercalcemia Type 3 (FHH3) Narrows Localization to a &lt;3.5 Megabase Pair Region on Chromosome 19q13.3Semantic proximity Read ↗

Familial benign hypocalciuric hypercalcaemia.Semantic proximity Read ↗

Genetic linkage analysis in familial benign hypercalcemia using a candidate gene strategy. I. Studies in four families.Semantic proximity Read ↗

Congenital Hyperphosphatemic Conditions Caused by the Deficient Activity of FGF23.Semantic proximity Read ↗

Familial Hypocalciuric Hypercalcemia: The Challenge of DiagnosisSemantic proximity Read ↗

Non-Classical Presentations of Rare Hereditary Hypoparathyroidism: A Case Series of CASR, GNA11 and GATA3 Mutations.Semantic proximity Read ↗

Hyperphosphatemic tumoral calcinosis caused by FGF23 compound heterozygous mutations: what are the therapeutic options for a better control of phosphatemia?Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.