Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Aging normal and dystrophic mouse muscle: Analysis of myogenicity in cultures of living single fibers

1 Feb 1998

Abstract excerpt

In the early stages of Duchenne muscular dystrophy, chronic muscle degeneration is counterbalanced by regeneration whose progressive failure beyond the fifth year is attributed to an accelerated senescence associated with excessive myogenic cell division. This idea was tested by counting the numbers of myogenic cells accumulating over 90 h around individual living fibers isolated from muscles of dystrophic (mdx)...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Exercise and Physiological Responses
Share to Muscle Physiology and Disorders

Source

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Identifiers

PMID: 9466592
DOI: 10.1002/(sici)1097-4598(199802)21:2<173::aid-mus4>3.0.co;2-8

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Aging normal and dystrophic mouse muscle: Analysis of myogenicity in cultures of living single fibersPMID 9466592Read ↗

Replicative aging down‐regulates the myogenic regulatory factors in human myoblastsSemantic proximity Read ↗

Cell culture systems as tools for studying age-related changes in skeletal muscle.Semantic proximity Read ↗

Rapid depletion of muscle progenitor cells in dystrophic mdx/utrophin−/− miceSemantic proximity Read ↗

Cellular senescence in human myoblasts is overcome by human telomerase reverse transcriptase and cyclin‐dependent kinase 4: consequences in aging muscle and therapeutic strategies for muscular dystrophiesSemantic proximity Read ↗

Immortalized pathological human myoblasts: towards a universal tool for the study of neuromuscular disordersSemantic proximity Read ↗

Differentiation rather than aging of muscle stem cells abolishes their telomerase activitySemantic proximity Read ↗

Primary human muscle precursor cells obtained from young and old donors produce similar proliferative, differentiation and senescent profiles in cultureSemantic proximity Read ↗

The mitotic clock in skeletal muscle regeneration, disease and cell mediated gene therapySemantic proximity Read ↗

Re-expression of cell cycle markers in aged neurons and muscles: Whether cells should divide or die?Semantic proximity Read ↗

Lifespan analysis of dystrophic <i>mdx</i> fast-twitch muscle morphology and its impact on contractile functionSemantic proximity Read ↗

Retarded myogenic cell replication in regenerating skeletal muscles of old mice: an autoradiographic study in young and old BALBc and SJL/J miceSemantic proximity Read ↗

Bioengineered 3D Skeletal Muscle Model Reveals Complement 4b as a Cell‐Autonomous Mechanism of Impaired Regeneration with AgingSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.