Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Plasma Activity and Insertion/Deletion Polymorphism of Angiotensin I–Converting Enzyme

20 Jan 1998

Abstract excerpt

BACKGROUND: Tissue proliferation is almost invariably observed in recurrent lesions within stents, and ACE, a factor of smooth muscle cell proliferation, may play an important role. Plasma ACE level is largely controlled by the insertion/deletion (I/D) polymorphism of the enzyme gene. The association among restenosis within coronary stents, plasma ACE level, and the I/D polymorphism is analyzed in the present...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Blood Coagulation and Thrombosis Mechanisms
Share to Coagulation, Bradykinin, Polyphosphates, and Angioedema
Share to Renin-Angiotensin System Studies

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 9445166
DOI: 10.1161/01.cir.97.2.147

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Plasma Activity and Insertion/Deletion Polymorphism of Angiotensin I–Converting EnzymePMID 9445166Read ↗

Relation between the insertion/deletion polymorphism of the angiotensin I converting enzyme gene and restenosis after coronary stenting.Semantic proximity Read ↗

D allele of the angiotensin I-converting enzyme is a major risk factor for restenosis after coronary stenting.Semantic proximity Read ↗

[D/D genotype of the gene for angiotensin converting enzyme as a risk factor for post-stent coronary restenosis].Semantic proximity Read ↗

Angiotensin-converting enzyme insertion/deletion polymorphism and risk of restenosis after directional coronary atherectomy followed by stent implantation.Semantic proximity Read ↗

Renin-angiotensin-aldosterone system (RAAS) gene polymorphism as a risk factor of coronary in-stent restenosis.Semantic proximity Read ↗

Angiotensin I-converting enzyme (ACE) inhibitors and restenosis after coronary artery stenting in patients with the DD genotype of the ACE gene.Semantic proximity Read ↗

Association study of the I/D polymorphism and plasma angiotensin-converting enzyme (ACE) as risk factors for stent restenosis.Semantic proximity Read ↗

Association between the ACE genotype and coronary artery disease. Insights from studies on restenosis, vasomotion and thrombosis.Semantic proximity Read ↗

Association ofACE Gene Polymorphisms with In-Stent Restenosis by Stent Type (Biomime, Supraflex, Xience)Semantic proximity Read ↗

Effect of ACE inhibitors on angiographic restenosis after coronary stenting (PARIS): a randomised, double-blind, placebo-controlled trial.Semantic proximity Read ↗

Association of seven renin angiotensin system gene polymorphisms with restenosis in patients following coronary stenting.Semantic proximity Read ↗

Association of ACE insertion or deletion polymorphisms with the risk of coronary restenosis after percutaneous coronary intervention: A meta-analysis.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.