Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Evidence for the polyclonal nature of focal nodular hyperplasia of the liver by the study of X-chromosome inactivation

1 Oct 1997

Abstract excerpt

Focal nodular hyperplasia (FNH) of the liver is a benign tumor commonly considered as a reactive disorder related to a pre-existing vascular malformation. However, the pathogenesis of this lesion has been recently discussed. To determine whether FNH is a polyclonal or a clonal lesion, we investigated the inactivation pattern of the X chromosome, using molecular genetic analysis of the DNA methylation pattern at a...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cholangiocarcinoma and Gallbladder Cancer Studies
Share to Genetic and Kidney Cyst Diseases
Share to Histiocytic Disorders and Treatments

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 9328310
DOI: 10.1002/hep.510260414

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Evidence for the polyclonal nature of focal nodular hyperplasia of the liver by the study of X-chromosome inactivationPMID 9328310Read ↗

Use of X-chromosome inactivation pattern and laser microdissection to determine the clonal origin of focal nodular hyperplasia of the liver.Semantic proximity Read ↗

Clonality and allelotype analyses of focal nodular hyperplasia compared with hepatocellular adenoma and carcinoma.Semantic proximity Read ↗

Molecular pathogenesis of focal nodular hyperplasia and hepatocellular adenoma.Semantic proximity Read ↗

Clinical, morphologic, and molecular features defining so-called telangiectatic focal nodular hyperplasias of the liver.Semantic proximity Read ↗

Dermatofibroma is a clonal proliferative disease.Semantic proximity Read ↗

Chromosomal analysis of hepatic adenoma and focal nodular hyperplasia by comparative genomic hybridizationSemantic proximity Read ↗

Desmoid fibromatosis is a clonal process.Semantic proximity Read ↗

Monoclonality in normal epithelium and in hyperplastic and neoplastic lesions of the breastSemantic proximity Read ↗

Desmoid Tumor Is a Clonal Cellular Proliferation: PCR Amplification of HUMARA for Analysis of Patterns of X-Chromosome InactivationSemantic proximity Read ↗

Clonal analysis of cutaneous fibrous histiocytoma (dermatofibroma)Semantic proximity Read ↗

Clonal Nature of Sclerosing Polycystic Adenosis of Salivary Glands Demonstrated by Using the Polymorphism of the Human Androgen Receptor (HUMARA) Locus as a MarkerSemantic proximity Read ↗

Polyclonal nature of diffuse proliferation of interstitial cells of Cajal in patients with familial and multiple gastrointestinal stromal tumoursSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.