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Article

Accelerated loss of telomeric repeats may not explain accelerated replicative decline of Werner syndrome cells

1 Jun 1996

Abstract excerpt

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Topics

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Share to Chromosomal and Genetic Variations
Share to DNA Repair Mechanisms
Share to Telomeres, Telomerase, and Senescence

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 8641691
DOI: 10.1007/bf02346184

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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Accelerated loss of telomeric repeats may not explain accelerated replicative decline of Werner syndrome cellsPMID 8641691Read ↗

Accelerated loss of telomeric repeats may not explain accelerated replicative decline of Werner syndrome cellsSemantic proximity Read ↗

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Telomerase prevents the accelerated cell ageing of Werner syndrome fibroblastsSemantic proximity Read ↗

Essential role of limiting telomeres in the pathogenesis of Werner syndromeSemantic proximity Read ↗

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Telomere ResQue and preservation—Roles for the Werner syndrome protein and other RecQ helicasesSemantic proximity Read ↗

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Accelerated Telomere Shortening in the Human Inactive X ChromosomeSemantic proximity Read ↗

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Rescue of a telomere length defect of Nijmegen breakage syndrome cells requires NBS and telomerase catalytic subunitSemantic proximity Read ↗

Release of Telomeric DNA from Chromosomes in Immortal Human Cells Lacking Telomerase ActivitySemantic proximity Read ↗

Abnormal telomere dynamics of B-lymphoblastoid cell strains from Werner's syndrome patients transformed by Epstein – Barr virusSemantic proximity Read ↗

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