Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Striatal glucose consumption in chorea-free subjects at risk of Huntington's disease.

Journal of neurology - 1 Nov 1993

Kuwert T, Lange H W, Boecker H, Titz H, Herzog H, Aulich A, Wang B C, Nayak U, Feinendegen L E

Abstract excerpt

Controversial data have been reported with regard to the diagnostic value of the positron emission tomographic (PET) measurement of striatal glucose consumption (rCMRGlc) in chorea-free subjects at risk of Huntington's disease (HD). For further clarification of this issue we measured striatal and cerebellar rCMRGlc in 27 chorea-free subjects at risk of HD, 20 patients with manifest HD and 20 control subjects,...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Adolescent
Share to Corpus Striatum
Share to Disease Susceptibility

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Genetic Predisposition to Disease

Share to Glucose

Share to Heterozygote

Share to Huntington Disease

Source

Identifiers

PMID: 8138819
DOI: 10.1007/BF00870669

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 8 closest

Striatal glucose consumption in chorea-free subjects at risk of Huntington's disease.PMID 8138819Read ↗

A comparison of neurological, metabolic, structural, and genetic evaluations in persons at risk for Huntington's disease.Semantic proximity Read ↗

Striatal glucose metabolism and dopamine D2 receptor binding in asymptomatic gene carriers and patients with Huntington's disease.Semantic proximity Read ↗

Brain glucose metabolism in adults with ataxia-telangiectasia and their asymptomatic relatives.Semantic proximity Read ↗

Striatal D1 and D2 receptor binding in patients with Huntington's disease and other choreas. A PET study.Semantic proximity Read ↗

Decreased Metabolism in the Cerebral Cortex in Early-Stage Huntington's Disease: A Possible Biomarker of Disease Progression?Semantic proximity Read ↗

Metabolic and Volumetric Alterations in the Basal Ganglia and the Cerebellum in Dopa-Responsive Dystonia in Symptomatic and Asymptomatic GCH1 Mutation Carriers.Semantic proximity Read ↗

Reduced basal ganglia blood flow and volume in pre-symptomatic, gene-tested persons at-risk for Huntington's disease.Semantic proximity Read ↗

Bioenergetic dysregulation in the basal ganglia and cerebellum of patients with premanifest and manifest Huntington’s diseaseSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.