Article
Canonical ETI-Dependent and -Independent Pathways Mediate Autoimmunity Caused by Loss of CBP60b Clade Function.
Molecular plant pathology - 1 Jul 2026
Li Lu-Shen, Yang Yan-Yan, Ma Lin, Wang Shu-Lei, Li Sha
Abstract excerpt
Calmodulin-Binding Protein 60 (CBP60), a plant-specific atypical transcription factor family, plays pivotal roles in reprogramming plant immunity. Loss of function of the CBP60b and CBP60b/c/d/e/f subfamily constitutively activates immune responses (autoimmunity). However, the specific mechanisms and pathways underlying this autoimmunity remain elusive. Here, through EMS mutagenesis of the cbp60b mutant, we...
Read the complete abstract on PubMedTopics
Share this publication in a Topic to start or enrich a Post.
