Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

R79E Mutation in the Nicotinic Acetylcholine Receptor β1 Subunit Drives High-Level Resistance to Neonicotinoid Insecticides in Bemisia tabaci.

Journal of agricultural and food chemistry - 29 Jul 2026

Li Kaixin, Wu Ningning, Hu Jinghao, Hu Xichao, Tang Haoyu, Xu Wei, Dong Ziqi, Lai Xiaodong, Xu Lihan, Fu Chongzhen, Liu Haoran, Tan Huiwen, Geng Ziqiong, Liu Xiaolan, Ma Sanyuan, Zhang Yongjun, Guo Lei

Abstract excerpt

The whitefly, Bemisia tabaci, a globally significant pest transmitting over 200 plant viruses, has developed substantial resistance to insecticides, particularly neonicotinoids. Using near-isogenic lines, bulked segregant analysis (BSA), and genetic linkage analysis, we identified a major resistance locus on chromosome 6. The locus harbored dual mutations in the nicotinic acetylcholine receptor (nAChR) β1 subunit...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Hemiptera
Share to Insect Proteins

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Insecticide Resistance

Share to Insecticides

Share to Mutation

Share to Receptors, Nicotinic

Source

Identifiers

PMID: 42334210
DOI: 10.1021/acs.jafc.6c03658

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 18 Oct 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

R79E Mutation in the Nicotinic Acetylcholine Receptor β1 Subunit Drives High-Level Resistance to Neonicotinoid Insecticides in Bemisia tabaci.PMID 42334210Read ↗

Dual mutations in the whitefly nicotinic acetylcholine receptor β1 subunit confer target-site resistance to multiple neonicotinoid insecticides.Semantic proximity Read ↗

Mutations in the Bemisia tabaci para sodium channel gene associated with resistance to a pyrethroid plus organophosphate mixture.Semantic proximity Read ↗

V101I and R81T mutations in the nicotinic acetylcholine receptor β1 subunit are associated with neonicotinoid resistance in Myzus persicae.Semantic proximity Read ↗

Lack of cross-resistance between neonicotinoids and sulfoxaflor in field strains of Q-biotype of whitefly, Bemisia tabaci, from eastern China.Semantic proximity Read ↗

The relative contribution of target-site mutations in complex acaricide resistant phenotypes as assessed by marker assisted backcrossing in Tetranychus urticae.Semantic proximity Read ↗

Transcription analysis of neonicotinoid resistance in Mediterranean (MED) populations of B. tabaci reveal novel cytochrome P450s, but no nAChR mutations associated with the phenotype.Semantic proximity Read ↗

Evolutionary trade-offs of insecticide resistance - The fitness costs associated with target-site mutations in the nAChR of Drosophila melanogaster.Semantic proximity Read ↗

Global distribution and origin of target site insecticide resistance mutations in Tetranychus urticae.Semantic proximity Read ↗

Insecticide resistance and characteristics of mutations related to target site insensitivity of diamondback moths in Taiwan.Semantic proximity Read ↗

Multiple origins of pyrethroid resistance in sympatric biotypes of Bemisia tabaci (Hemiptera: Aleyrodidae).Semantic proximity Read ↗

Mutations in the sodium channel associated with pyrethroid resistance in the greenhouse whitefly, Trialeurodes vaporariorum.Semantic proximity Read ↗

Development of a low-density DNA microarray for diagnosis of target-site mutations of pyrethroid and organophosphate resistance mutations in the whitefly Bemisia tabaci.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.