Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Dysregulation of lncRNA GATA3-AS1 is Involved in the Pathogenesis of Pulpitis by the Regulation of miR-17-3p

1 Mar 2025

Abstract excerpt

Purpose: When the pulp is inflamed or injured, cell morphology, gene expression, and synaptic connections change occur in the medullary dorsal horn, causing inflammation pain and formatting the pulpitis pain. To examine the impact of lncRNA GATA3-AS1 regulation of miR-17-3p on bioactivity and inflammation of lipopolysaccharides (LPS)-stimulated human dental pulp stem cells (hDPSCs). Patients and Methods: The...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Bone Metabolism and Diseases
Share to Endodontics and Root Canal Treatments
Share to Pain Mechanisms and Treatments

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 40162083
DOI: 10.2147/jir.s504048

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Dysregulation of lncRNA GATA3-AS1 is Involved in the Pathogenesis of Pulpitis by the Regulation of miR-17-3pPMID 40162083Read ↗

Identification of key genes involved in neuroendocrine regulation in pulpitis: bioinformatics and experimental analysisSemantic proximity Read ↗

Stathmin regulates the proliferation and differentiation of inflammatory hDPSCs through canonical Wnt/β-catenin pathwaySemantic proximity Read ↗

ATF3 triggers M2 macrophage polarization to protect against pulp inflammation through WNT4 regulationSemantic proximity Read ↗

Osteopontin interacts with dendritic cells and macrophages in pulp inflammation: Comprehensive transcriptomic analysis and laboratory investigationsSemantic proximity Read ↗

Combined Transcriptomic and Proteomic Profiling of the Mouse Anterior Cingulate Cortex Identifies Potential Therapeutic Targets for Pulpitis-Induced PainSemantic proximity Read ↗

Single-cell Resolution Spatial Transcriptomics Delineates the Inflammatory Landscape of Human Dental Pulp: Regional Crosstalk and Therapeutic ImplicationsSemantic proximity Read ↗

Effects of Porphyromonas gingivalis lipopolysaccharide on the behavior of human dental pulp stem cells in vitro and their inflammation-related transcriptomics profileSemantic proximity Read ↗

Identification of Pulpitis-Related Potential Biomarkers Using Bioinformatics ApproachSemantic proximity Read ↗

Epigenetic regulation in dental pulp inflammationSemantic proximity Read ↗

Overexpression of MicroRNA ‐155 aggravates pulpitis by targeting kinesin superfamily Proteins‐5C based on illumina high‐throughput sequencingSemantic proximity Read ↗

Effects of miR-629-5p on odontoblast differentiation and target gene TEAD4 expression of dental pulp stem cellsSemantic proximity Read ↗

MicroRNA-27a-5p downregulates the expression of proinflammatory cytokines in lipopolysaccharide-stimulated human dental pulp cells via NF-κB signaling pathwaySemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.