Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

CD74 + fibroblasts proliferate upon mechanical stretching to promote angiogenesis in keloids

14 Oct 2024

Abstract excerpt

Abstract The healing of human skin wounds is susceptible to perturbation caused by excessive mechanical stretching, resulting in enlarged scars, hypertrophic scars, or even keloids in predisposed individuals. Keloids are fibro‐proliferative scar tissues that extend beyond the initial wound boundary, consisting of the actively progressing periphery and the quiescent center. The stretch‐associated outgrowth and...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Dermatologic Treatments and Research
Share to Tissue Engineering and Regenerative Medicine
Share to Wound Healing and Treatments

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 39400419
DOI: 10.1096/fj.202401302r

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

CD74 + fibroblasts proliferate upon mechanical stretching to promote angiogenesis in keloidsPMID 39400419Read ↗

New Anti‐Fibrotic Strategies for Keloids: Insights From Single‐Cell Multi‐OmicsSemantic proximity Read ↗

Single cell transcriptomics reveals the cellular heterogeneity of keloids and the mechanism of their aggressivenessSemantic proximity Read ↗

Spatiotemporal Single-Cell Roadmap of Human Skin Wound HealingSemantic proximity Read ↗

Decoding the molecular landscape of keloids: new insights from single-cell transcriptomicsSemantic proximity Read ↗

Spatiotemporal single-cell roadmap of human skin wound healingSemantic proximity Read ↗

Keloid disorder: Fibroblast differentiation and gene expression profile in fibrotic skin diseasesSemantic proximity Read ↗

Single‐cell RNA sequencing highlights the role of proinflammatory fibroblasts, vascular endothelial cells, and immune cells in the keloid immune microenvironmentSemantic proximity Read ↗

Single-cell RNA-seq reveals fibroblast heterogeneity and increased mesenchymal fibroblasts in human fibrotic skin diseasesSemantic proximity Read ↗

Transcriptome Profiling Reveals Important Transcription Factors and Biological Processes in Skin Regeneration Mediated by Mechanical StretchSemantic proximity Read ↗

Longitudinal <i>in vivo</i> human wound healing model defines key role for smooth muscle cells in ECM remodelingSemantic proximity Read ↗

An advanced ADSC therapy for keloid prevention using identification of functional subgroups by single cell transcriptomic analysisSemantic proximity Read ↗

Revealing the pathogenesis of keloids based on the status: Active vs inactiveSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.