Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Epstein-Barr virus suppresses N6-methyladenosine modification of TLR9 to promote immune evasion

25 Mar 2024

Abstract excerpt

Epstein-Barr virus (EBV) is a human tumor virus associated with a variety of malignancies, including nasopharyngeal carcinoma, gastric cancers, and B-cell lymphomas. N 6 -methyladenosine (m 6 A) modifications modulate a wide range of cellular processes and participate in the regulation of virus-host cell interactions. Here, we discovered that EBV infection downregulates Toll-like receptor 9 (TLR9) m 6 A...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cytomegalovirus and herpesvirus research
Share to RNA modifications and cancer
Share to Viral-associated cancers and disorders

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 38537697
DOI: 10.1016/j.jbc.2024.107226

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Epstein-Barr virus suppresses N6-methyladenosine modification of TLR9 to promote immune evasionPMID 38537697Read ↗

RNA m6A methylation regulates virus–host interaction and EBNA2 expression during Epstein–Barr virus infectionSemantic proximity Read ↗

N(6)-methyladenosine-binding protein YTHDF1 suppresses EBV replication and promotes EBV RNA decaySemantic proximity Read ↗

Epstein-Barr virus-encoded microRNAs as regulators in host immune responsesSemantic proximity Read ↗

The Role of Epstein-Barr Virus in Modulating Key Tumor Suppressor Genes in Associated Malignancies: Epigenetics, Transcriptional, and Post-Translational ModificationsSemantic proximity Read ↗

EBV Lytic-Phase Protein BGLF5 Contributes to TLR9 Downregulation during Productive InfectionSemantic proximity Read ↗

Multiple viral microRNAs regulate interferon release and signaling early during infection with Epstein-Barr virusSemantic proximity Read ↗

Targeting the signaling in Epstein–Barr virus-associated diseases: mechanism, regulation, and clinical studySemantic proximity Read ↗

miRNAs: EBV Mechanism for Escaping Host’s Immune Response and Supporting TumorigenesisSemantic proximity Read ↗

N6-methyladenosine promotes induction of ADAR1-mediated A-to-I RNA editing to suppress aberrant antiviral innate immune responsesSemantic proximity Read ↗

An update: Epstein-Barr virus and immune evasion via microRNA regulationSemantic proximity Read ↗

TRIM29 promotes DNA virus infections by inhibiting innate immune responseSemantic proximity Read ↗

Epigenetic Mechanisms in Latent Epstein-Barr Virus Infection and Associated CancersSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.