Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Exercise combined with postbiotics treatment results in synergistic improvement of mitochondrial function in the brain of male transgenic mice for Alzheimer’s disease

18 Dec 2023

Abstract excerpt

Abstract Background It has been suggested that exercise training and postbiotic supplement could decelerate the progress of functional and biochemical deterioration in double transgenic mice overexpresses mutated forms of the genes for human amyloid precursor protein (APP sw ) and presenilin 1 (m146L) (APP/PS1 TG ). Our earlier published data indicated that the mice performed better than controls on the Morris...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Alzheimer's disease research and treatments
Share to Medicinal Plants and Neuroprotection
Share to Tryptophan and brain disorders

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 38110905
DOI: 10.1186/s12868-023-00836-x

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Exercise combined with postbiotics treatment results in synergistic improvement of mitochondrial function in the brain of male transgenic mice for Alzheimer’s diseasePMID 38110905Read ↗

Exercise combined with postbiotics treatment results in synergistic improvement of mitochondrial function in the brain of male transgenic mice for Alzheimer’s diseaseSemantic proximity Read ↗

Chronic administration of prebiotics and probiotics ameliorates pathophysiological hallmarks of Alzheimer’s disease in a APP/PS1 transgenic mouse modelSemantic proximity Read ↗

Exercise and probiotics attenuate the development of Alzheimer's disease in transgenic mice: Role of microbiomeSemantic proximity Read ↗

Physical activity delays hippocampal neurodegeneration and rescues memory deficits in an Alzheimer disease mouse modelSemantic proximity Read ↗

Role of the Nrf2/ARE downregulation and mtDNA damage in the cognitive impairments in APP/PS1 miceSemantic proximity Read ↗

BHBA treatment improves cognitive function by targeting pleiotropic mechanisms in transgenic mouse model of Alzheimer's diseaseSemantic proximity Read ↗

Gut Flora-Targeted Photobiomodulation Therapy Improves Senile Dementia in an Aß-Induced Alzheimer’s Disease Animal ModelSemantic proximity Read ↗

Pharmacological intervention in a transgenic mouse model improves Alzheimer's-associated pathological phenotype: Involvement of proteasome activation.Semantic proximity Read ↗

Long-term caffeine treatment of Alzheimer mouse models ameliorates behavioural deficits and neuron loss and promotes cellular and molecular markers of neurogenesisSemantic proximity Read ↗

Maternal Resveratrol Supplementation Prevents Cognitive Decline in Senescent Mice OffspringSemantic proximity Read ↗

Orally active mGluR2/3 metabotropic antagonist pro-drug mimics the beneficial effects of physical exercise on neurogenesis, behavior, and exercise-related molecular pathways in an Alzheimer’s disease mouse modelSemantic proximity Read ↗

Neurogenesis and neuroprotection in postischemic brain neurodegeneration with Alzheimer phenotype: is there a role for curcumin?Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.