Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Quantitative and causal analysis for inflammatory genes and the risk of Parkinson's disease.

Frontiers in immunology - 1 Jan 2023

Yi Minhan, Li Jiaxin, Jian Shijie, Li Binbin, Huang Zini, Shu Li, Zhang Yuan

Abstract excerpt

Background: The dysfunction of immune system and inflammation contribute to the Parkinson's disease (PD) pathogenesis. Cytokines, oxidative stress, neurotoxin and metabolism associated enzymes participate in neuroinflammation in PD and the genes involved in them have been reported to be associated with the risk of PD. In our study, we performed a quantitative and causal analysis of the relationship between...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to ADP-ribosyl Cyclase
Share to Arylamine N-Acetyltransferase

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Aryldialkylphosphatase

Share to Cytochrome P-450 CYP2D6

Share to Genetic Predisposition to Disease

Share to Interleukin-6

Share to Parkinson Disease

Share to Tumor Necrosis Factor-alpha

Source

Open in PubMed Open full text

Identifiers

PMID: 36926335
DOI: 10.3389/fimmu.2023.1119315
PMCID: PMC10011457

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 3 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Quantitative and causal analysis for inflammatory genes and the risk of Parkinson's disease.PMID 36926335Read ↗

Inflammation-related gene polymorphisms associated with Parkinson’s disease: an updated meta-analysisSemantic proximity Read ↗

Genetic variability of inflammation and oxidative stress genes does not play a major role in the occurrence of adverse events of dopaminergic treatment in Parkinson’s diseaseSemantic proximity Read ↗

Circulating inflammatory proteins and risk of Parkinson’s disease and other neurodegenerative disorders: a two-sample Mendelian randomization studySemantic proximity Read ↗

A Mendelian randomization study investigating the causal role of inflammation on Parkinson's disease.Semantic proximity Read ↗

Circulating inflammatory proteins and risk of Parkinson’s disease and other neurodegenerative disorders: a two-sample Mendelian randomization studySemantic proximity Read ↗

The causal relationship between Parkinson’s disease and inflammatory cytokines: a bidirectional two-sample Mendelian randomization studySemantic proximity Read ↗

Genetically predicted levels of circulating cytokines and risk of Alzheimer's and Parkinson's disease: evidence from complementary genetic methodsSemantic proximity Read ↗

Key Non-coding Variants in Three Neuroapoptosis and Neuroinflammation-Related LncRNAs Are Protectively Associated with Susceptibility to Parkinson's Disease and Some of Its Clinical Features.Semantic proximity Read ↗

Association Between Neuroinflammation and Parkinson's Disease: A Comprehensive Mendelian Randomization Study.Semantic proximity Read ↗

Causal association of circulating inflammatory proteins on neurodegenerative diseases: Insights from a mendelian randomization study.Semantic proximity Read ↗

Assessing causal relationship between circulating cytokines and age-related neurodegenerative diseases: a bidirectional two-sample Mendelian randomization analysisSemantic proximity Read ↗

Assessment of causal factors for Parkinson's disease in European populations: A phenome-wide Mendelian randomisation analysis.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.