Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Relationship between Glucose-6-Phosphate Dehydrogenase Deficiency, X-Chromosome Inactivation and Inflammatory Markers

31 Jan 2023

Abstract excerpt

Recent studies suggest that X-linked glucose-6-phosphate dehydrogenase (G6PD) deficiency entails a proinflammatory state that may increase the risk of several disease conditions. However, it is not clear how this relates to the degree of enzyme insufficiency and, in heterozygous females, to skewed inactivation of the X chromosome. This study aimed to (i) investigate the enzyme activity in a cohort of 232 subjects...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Blood groups and transfusion
Share to Hemoglobinopathies and Related Disorders
Share to Neonatal Health and Biochemistry

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 36829893
DOI: 10.3390/antiox12020334

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 3 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Relationship between Glucose-6-Phosphate Dehydrogenase Deficiency, X-Chromosome Inactivation and Inflammatory MarkersPMID 36829893Read ↗

Glucose-6-Phosphate Dehydrogenase Deficiency: Disadvantages and Possible BenefitsSemantic proximity Read ↗

Glucose-6-phosphate dehydrogenase deficiencySemantic proximity Read ↗

Glucose-6-phosphate dehydrogenase--from oxidative stress to cellular functions and degenerative diseases.Semantic proximity Read ↗

DNA hypermethylation and X chromosome inactivation are major determinants of phenotypic variation in women heterozygous for G6PD mutations.Semantic proximity Read ↗

Molecular basis and enzymatic properties of glucose 6-phosphate dehydrogenase volendam, leading to chronic nonspherocytic anemia, granulocyte dysfunction, and increased susceptibility to infections.Semantic proximity Read ↗

Serum microRNAs as new biomarkers for detecting subclinical hemolysis in the nonacute phase of G6PD deficiencySemantic proximity Read ↗

Glucose-6-phosphate dehydrogenase deficiency in Italian blood donors: prevalence and molecular defect characterization.Semantic proximity Read ↗

Epidemiological shift of glucose-6-phosphate dehydrogenase mutations in northern Italy in the last 15 years.Semantic proximity Read ↗

Treatment strategies for glucose-6-phosphate dehydrogenase deficiency: past and future perspectives.Semantic proximity Read ↗

G6PD deficiency: the genotype-phenotype association.Semantic proximity Read ↗

Glucose-6-phosphate dehydrogenase deficiency in female octogenarians, nanogenarians, and centenarians.Semantic proximity Read ↗

Hemolytic anemias due to erythrocyte enzyme deficiencies.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.