Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Bisphenol A at a human exposed level can promote epithelial-mesenchymal transition in papillary thyroid carcinoma harbouring BRAFV600E mutation.

Journal of cellular and molecular medicine - 1 Feb 2021

Li Liuli, Li Hao, Zhang Jun, Gao Xin, Jin Hao, Liu Renqi, Zhang Zhen, Zhang Xuan, Wang Xichang, Qu Peng, Zhao Yuejiao, Lu Xiaobo

Abstract excerpt

Bisphenol A (BPA), a ubiquitous endocrine-disrupting chemical, alters the function of endocrine system and enhances the susceptibility to tumorigenesis in several hormone-dependent tumours as thyroid carcinoma. About 50% of papillary thyroid cancers (PTC), the most common type of thyroid malignancy, harbours the BRAFV600E mutation. This study aimed to investigate a potential combined effect of BPA exposure and...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Air Pollutants, Occupational
Share to Benzhydryl Compounds

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Cell Movement

Share to Epithelial-Mesenchymal Transition

Share to MAP Kinase Signaling System

Share to Phenols

Share to Proto-Oncogene Proteins B-raf

Share to Thyroid Cancer, Papillary

Source

Open in PubMed Open full text

Identifiers

PMID: 33469997
DOI: 10.1111/jcmm.16279
PMCID: PMC7875916

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Bisphenol A at a human exposed level can promote epithelial-mesenchymal transition in papillary thyroid carcinoma harbouring BRAFV600E mutation.PMID 33469997Read ↗

Differential Effects of Bisphenol A on Thyroid Cell Viability, Proliferation, and Migration Are Influenced by Genetic BackgroundSemantic proximity Read ↗

Genetic Background Dictates the Dichotomous Effects of Bisphenol A on Thyroid Cell Viability, Proliferation, and MigrationSemantic proximity Read ↗

A constitutive active MAPK/ERK pathway due to BRAFV600E positively regulates AHR pathway in PTC.Semantic proximity Read ↗

Exposure to flame retardant chemicals and occurrence and severity of papillary thyroid cancer: A case-control study.Semantic proximity Read ↗

Decabromodiphenyl ether exposure reduces dabrafenib sensitivity of papillary thyroid carcinoma harboring BRAFV600E mutation through the EGFR-CRAF-MAPK pathway: An in vitro study.Semantic proximity Read ↗

BRAFV600E and Microenvironment in Thyroid Cancer: A Functional Link to Drive Cancer ProgressionSemantic proximity Read ↗

Identification of serum metabolites associated with polybrominated diphenyl ethers (PBDEs) exposure in papillary thyroid carcinoma: A case–control studySemantic proximity Read ↗

The chemical environmental pollutants BPA and BPS induce alterations of the proteomic profile of different phenotypes of human breast cancer cells: A proposed interactome.Semantic proximity Read ↗

Association Between Obesity and BRAFV600E Mutation Status in Patients with Papillary Thyroid Cancer.Semantic proximity Read ↗

Low Dose of Bisphenol A Modulates Ovarian Cancer Gene Expression Profile and Promotes Epithelial to Mesenchymal Transition Via Canonical Wnt PathwaySemantic proximity Read ↗

An Animal Model Further Uncovers the Role of Mutant BrafV600E during Papillary Thyroid Cancer Development.Semantic proximity Read ↗

BRAF Inhibitors Induce Feedback Activation of RAS Pathway in Thyroid Cancer Cells.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.