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Article

Novel IL36RN Mutation Identified in Pediatric-Onset Generalized Pustular Psoriasis Causes IL36 Antagonist Degradation

7 Jan 2021

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Share to Autoimmune Bullous Skin Diseases
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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 33415665
DOI: 10.1007/s10875-020-00944-x

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Novel IL36RN Mutation Identified in Pediatric-Onset Generalized Pustular Psoriasis Causes IL36 Antagonist DegradationPMID 33415665Read ↗

IL36RN mutations define a severe autoinflammatory phenotype of generalized pustular psoriasisSemantic proximity Read ↗

Rare Pathogenic Variants in IL36RN Underlie a Spectrum of Psoriasis-Associated Pustular PhenotypesSemantic proximity Read ↗

Mutations in IL36RN in Patients with Generalized Pustular PsoriasisSemantic proximity Read ↗

A newly revealed IL36RN mutation in sibling cases complements our IL36RN mutation statistics for generalized pustular psoriasis.Semantic proximity Read ↗

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Interleukin-36 receptor antagonist deficiency patient with a novel mutation.Semantic proximity Read ↗

Annular pustular psoriasis with a heterozygous IL36RN mutation.Semantic proximity Read ↗

Generalized pustular psoriasis: immunological mechanisms, genetics, and emerging therapeutics.Semantic proximity Read ↗

A viewpoint on the genetic determinants of generalised pustular psoriasis.Semantic proximity Read ↗

Pathophysiology of generalized pustular psoriasis.Semantic proximity Read ↗

Mutations in IL36RN/IL1F5 are associated with the severe episodic inflammatory skin disease known as generalized pustular psoriasis.Semantic proximity Read ↗

Prevalent and Rare Mutations in IL-36RN Gene in Chinese Patients with Generalized Pustular Psoriasis and Psoriasis VulgarisSemantic proximity Read ↗

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