Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Investigation of glycaemic traits in psychiatric disorders using Mendelian randomisation revealed a causal relationship with anorexia nervosa.

Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology - 1 May 2021

Adams Danielle M, Reay William R, Geaghan Michael P, Cairns Murray J

Abstract excerpt

Data from observational studies have suggested an involvement of abnormal glycaemic regulation in the pathophysiology of psychiatric illness. This may be an attractive target for clinical intervention as glycaemia can be modulated by both lifestyle factors and pharmacological agents. However, observational studies are inherently confounded, and therefore, causal relationships cannot be reliably established. We...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Anorexia Nervosa
Share to Blood Glucose

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Genome-Wide Association Study

Share to Mendelian Randomization Analysis

Share to Polymorphism, Single Nucleotide

Share to Risk Factors

Source

Open in PubMed Open full text

Identifiers

PMID: 32920595
DOI: 10.1038/s41386-020-00847-w
PMCID: PMC8115098

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Investigation of glycaemic traits in psychiatric disorders using Mendelian randomisation revealed a causal relationship with anorexia nervosa.PMID 32920595Read ↗

Investigating the effect of glycaemic traits on the risk of psychiatric illness using Mendelian randomisationSemantic proximity Read ↗

Utilising genomic association data for causal inference in anorexia nervosa.Semantic proximity Read ↗

Genome-wide association study identifies eight risk loci and implicates metabo-psychiatric origins for anorexia nervosaSemantic proximity Read ↗

Significant Locus and Metabolic Genetic Correlations Revealed in Genome-Wide Association Study of Anorexia Nervosa.Semantic proximity Read ↗

Mendelian Randomization Analysis Identifies Inverse Causal Relationship between External Eating and Metabolic Phenotypes.Semantic proximity Read ↗

Contemporary views on the genetics of anorexia nervosa.Semantic proximity Read ↗

Dissecting the biology of feeding and eating disorders.Semantic proximity Read ↗

Genetic contributions to the etiology of anorexia nervosa: New perspectives in molecular diagnosis and treatment.Semantic proximity Read ↗

Genetic risk factors for eating disorders: an update and insights into pathophysiologySemantic proximity Read ↗

Molecular mechanisms underlying anorexia nervosa: focus on human gene association studies and systems controlling food intake.Semantic proximity Read ↗

Mendelian randomization analyses reveal novel drug targets for anorexia nervosa.Semantic proximity Read ↗

Comparing genome-wide significant and chemosensory variants as instruments for dietary patterns in Mendelian randomisationSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.