Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The expression of miR‐125b in Nrf2‐silenced A549 cells exposed to hyperoxia and its relationship with apoptosis

12 Nov 2019

Abstract excerpt

Bronchopulmonary dysplasia (BPD) is a chronic lung disease that affects the quality of life of infants. At present, premature exposure to hyperoxia for extended periods of time is believed to affect the development of lung tissue and vascularity, resulting in BPD. The oxidative stress caused by hyperoxia exposure is an important risk factor for BPD in premature infants. Nuclear factor E2-related factor 2 (Nrf2)...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Fatty Acid Research and Health
Share to MicroRNA in disease regulation
Share to Neonatal Respiratory Health Research

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 31713992
DOI: 10.1111/jcmm.14808

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 10 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The expression of miR‐125b in Nrf2‐silenced A549 cells exposed to hyperoxia and its relationship with apoptosisPMID 31713992Read ↗

Nesfatin-1 Suppresses Inflammation in Bronchopulmonary Dysplasia by Regulating HMGB-1/TLR4/p65/NLRP3 Signaling PathwaySemantic proximity Read ↗

Long non-coding RNA MALAT1 plays a protective role in bronchopulmonary dysplasia via the inhibition of apoptosis and interaction with the Keap1/Nrf2 signal pathwaySemantic proximity Read ↗

MicroRNA‐421 inhibition alleviates bronchopulmonary dysplasia in a mouse model via targeting Fgf10Semantic proximity Read ↗

MicroRNA-195-5p targets neurofibromin 2 to mediate hyperoxia-induced alveolar dysplasiaSemantic proximity Read ↗

Differential expression of long non‐coding RNAs in hyperoxia‐induced bronchopulmonary dysplasiaSemantic proximity Read ↗

MicroRNA-214 prevents pulmonary angiogenesis and alveolarization in neonatal rats with hyperoxia-mediated impairment of lung development by blocking PlGF-dependent STAT3 signaling pathwaySemantic proximity Read ↗

SIRT1-Related Signaling Pathways and Their Association With Bronchopulmonary DysplasiaSemantic proximity Read ↗

MicroRNA Expression Profile in Hyperoxia-Exposed Newborn Mice During the Development of Bronchopulmonary DysplasiaSemantic proximity Read ↗

BCL6 attenuates hyperoxia-induced lung injury by inhibiting NLRP3-mediated inflammation in fetal mouseSemantic proximity Read ↗

MicroRNA-214 Prevents Pulmonary Angiogenesis and Alveolarization in Rat Models with Bronchopulmonary Dysplasia via PlGF-Dependent STAT3 Signaling PathwaySemantic proximity Read ↗

Role of the Nrf2/HO-1 axis in bronchopulmonary dysplasia and hyperoxic lung injuriesSemantic proximity Read ↗

Umbilical cord blood–derived exosomes from healthy term pregnancies protect against hyperoxia‐induced lung injury in miceSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.