Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Functional Implications of LH/hCG Receptors in Pregnancy-Induced Cushing Syndrome

12 Jan 2017

Abstract excerpt

Elevated human choriogonadotropin (hCG) may stimulate aberrantly expressed luteinizing hormone (LH)/hCG receptor (LHCGR) in adrenal glands, resulting in pregnancy-induced bilateral macronodular adrenal hyperplasia and transient Cushing syndrome (CS). To determine the role of LHCGR in transient, pregnancy-induced CS. We investigated the functional implications of LHCGRs in a patient presenting, at a tertiary...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Growth Hormone and Insulin-like Growth Factors
Share to Hypothalamic control of reproductive hormones
Share to Pituitary Gland Disorders and Treatments

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 29264446
DOI: 10.1210/js.2016-1021

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Functional Implications of LH/hCG Receptors in Pregnancy-Induced Cushing SyndromePMID 29264446Read ↗

Update of Genetic and Molecular Causes of Adrenocortical Hyperplasias Causing Cushing Syndrome.Semantic proximity Read ↗

The mystery of transient pregnancy-induced cushing's syndrome: a case report and literature review highlighting GNAS somatic mutations and LHCGR overexpression.Semantic proximity Read ↗

Whole Genome Expression Profiling of Glucose-Dependent Insulinotropic Peptide (GIP)- and Adrenocorticotropin-Dependent Adrenal Hyperplasias Reveals Novel Targets for the Study of GIP-Dependent Cushing’s SyndromeSemantic proximity Read ↗

Aberrant expression of human luteinizing hormone receptor by adrenocortical cells is sufficient to provoke both hyperplasia and Cushing's syndrome features.Semantic proximity Read ↗

ACTH-independent Cushing's syndrome due to ectopic endocrinologically functional adrenal tissue caused by a GNAS heterozygous mutation: a rare case of McCune-Albright syndrome accompanied by central amenorrhea and hypothyroidism: a case report and literature review.Semantic proximity Read ↗

Rare correlation of somatic PRKACA mutations with pregnancy-associated aldosterone- and cortisol-producing adenomas: a case report and literature review.Semantic proximity Read ↗

Genetics of primary macronodular adrenal hyperplasia.Semantic proximity Read ↗

Luteinizing hormone and human chorionic gonadotropin: distinguishing unique physiologic roles.Semantic proximity Read ↗

Aberrant G-protein coupled hormone receptor in adrenal diseases.Semantic proximity Read ↗

Diagnosis and management of primary bilateral macronodular adrenal hyperplasiaSemantic proximity Read ↗

A Transcriptome-Wide Screen for mRNAs Enriched in Fetal Leydig Cells: CRHR1 Agonism Stimulates Rat and Mouse Fetal Testis SteroidogenesisSemantic proximity Read ↗

Systematic Analysis of G Protein-Coupled Receptor Gene Expression in Adrenocorticotropin-Independent Macronodular Adrenocortical Hyperplasia Identifies Novel Targets for Pharmacological Control of Adrenal Cushing’s SyndromeSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.