Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Eleven Telomere, Epigenetic Clock, and Biomarker-Composite Quantifications of Biological Aging: Do They Measure the Same Thing?

24 Oct 2017

Abstract excerpt

The geroscience hypothesis posits that therapies to slow biological processes of aging can prevent disease and extend healthy years of life. To test such "geroprotective" therapies in humans, outcome measures are needed that can assess extension of disease-free life span. This need has spurred development of different methods to quantify biological aging. But different methods have not been systematically...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Birth, Development, and Health
Share to Genetics, Aging, and Longevity in Model Organisms
Share to Telomeres, Telomerase, and Senescence

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 29149257
DOI: 10.1093/aje/kwx346

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Eleven Telomere, Epigenetic Clock, and Biomarker-Composite Quantifications of Biological Aging: Do They Measure the Same Thing?PMID 29149257Read ↗

Comprehensive Comparison of Sixteen Markers of Biological Aging: Cross-Sectional and Longitudinal Results from the Berlin Aging Study II (BASE-II)Semantic proximity Read ↗

Effect of Modifiable Lifestyle Factors on Biological AgingSemantic proximity Read ↗

When to Trust Epigenetic Clocks: Avoiding False Positives in Aging InterventionsSemantic proximity Read ↗

A systematic review of phenotypic and epigenetic clocks used for aging and mortality quantification in humans.Semantic proximity Read ↗

Critical review of aging clocks and factors that may influence the pace of agingSemantic proximity Read ↗

A toolkit for quantification of biological age from blood-chemistry and organ-function-test data: BioAgeSemantic proximity Read ↗

Association of 4 epigenetic clocks with measures of functional health, cognition, and all-cause mortality in The Irish Longitudinal Study on Ageing (TILDA)Semantic proximity Read ↗

Generations of epigenetic clocks and their links to socioeconomic status in the Health and Retirement StudySemantic proximity Read ↗

Does Modulation of an Epigenetic Clock Define a Geroprotector?Semantic proximity Read ↗

Epigenetic Clock and Leukocyte Telomere Length Are Associated with Vitamin D Status but not with Functional Assessments and Frailty in the Berlin Aging Study IISemantic proximity Read ↗

Reversal of epigenetic aging and immunosenescent trends in humansSemantic proximity Read ↗

Associations between exposure to adverse childhood experiences and biological aging: Evidence from the Canadian Longitudinal Study on AgingSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.