Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

GBF1 differentially regulates CAT2 and PAD4 transcription to promote pathogen defense in Arabidopsis thaliana.

The Plant journal : for cell and molecular biology - 1 Sept 2017

Giri Mrunmay K, Singh Nidhi, Banday Zeeshan Z, Singh Vijayata, Ram Hathi, Singh Deepjyoti, Chattopadhyay Sudip, Nandi Ashis K

Abstract excerpt

G-BOX BINDING FACTOR 1 (GBF1) influences light-regulated seedling development in Arabidopsis, and inhibits CATALASE 2 (CAT2) expression during senescence. CAT2 functions as a scavenger of hydrogen peroxide. The role of GBF1 in the defense response is not known. We report here that GBF1 positively influences the defense against virulent and avirulent strains of Pseudomonas syringae. The gbf1 mutants are...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Carboxylic Ester Hydrolases
Share to Disease Resistance

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Indoles

Share to Plant Diseases

Share to Plants, Genetically Modified

Share to Pseudomonas syringae

Share to Salicylic Acid

Share to Thiazoles

Source

Identifiers

PMID: 28622438
DOI: 10.1111/tpj.13608

Genes

GBF1

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

GBF1 differentially regulates CAT2 and PAD4 transcription to promote pathogen defense in Arabidopsis thaliana.PMID 28622438Read ↗

Arabidopsis GH3-LIKE DEFENSE GENE 1 is required for accumulation of salicylic acid, activation of defense responses and resistance to Pseudomonas syringae.Semantic proximity Read ↗

Overexpression of GbERF confers alteration of ethylene-responsive gene expression and enhanced resistance to Pseudomonas syringae in transgenic tobacco.Semantic proximity Read ↗

The Arabidopsis gene SIGMA FACTOR-BINDING PROTEIN 1 plays a role in the salicylate- and jasmonate-mediated defence responses.Semantic proximity Read ↗

The Arabidopsis thaliana At4g13040 gene, a unique member of the AP2/EREBP family, is a positive regulator for salicylic acid accumulation and basal defense against bacterial pathogens.Semantic proximity Read ↗

G-Box binding factor1 reduces CATALASE2 expression and regulates the onset of leaf senescence in Arabidopsis.Semantic proximity Read ↗

GeBP/GPL transcription factors regulate a subset of CPR5-dependent processes.Semantic proximity Read ↗

Arabidopsis clade I TGA factors regulate apoplastic defences against the bacterial pathogen Pseudomonas syringae through endoplasmic reticulum-based processes.Semantic proximity Read ↗

An important role of a BAHD acyl transferase-like protein in plant innate immunity.Semantic proximity Read ↗

Regulators of G‐protein signalling in Fusarium verticillioides mediate differential host–pathogen responses on nonviable versus viable maize kernelsSemantic proximity Read ↗

The Arabidopsis thaliana dihydroxyacetone phosphate reductase gene SUPPRESSSOR OF FATTY ACID DESATURASE DEFICIENCY1 is required for glycerolipid metabolism and for the activation of systemic acquired resistance.Semantic proximity Read ↗

Gac two-component system in Pseudomonas syringae pv. tabaci is required for virulence but not for hypersensitive reaction.Semantic proximity Read ↗

CPTF1, a CREB-like transcription factor, is involved in the oxidative stress response in the phytopathogen Claviceps purpurea and modulates ROS level in its host Secale cereale.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.