SLC2A3 single-nucleotide polymorphism and duplication influence cognitive processing and population-specific risk for attention-deficit/hyperactivity disorder.

doi:10.1111/jcpp.12702

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Article

SLC2A3 single-nucleotide polymorphism and duplication influence cognitive processing and population-specific risk for attention-deficit/hyperactivity disorder.

Journal of child psychology and psychiatry, and allied disciplines - 1 Jul 2017

Merker Sören, Reif Andreas, Ziegler Georg C, Weber Heike, Mayer Ute, Ehlis Ann-Christine, Conzelmann Annette, Johansson Stefan, Müller-Reible Clemens, Nanda Indrajit, Haaf Thomas, Ullmann Reinhard, Romanos Marcel, Fallgatter Andreas J, Pauli Paul, Strekalova Tatyana, Jansch Charline, Vasquez Alejandro Arias, Haavik Jan, Ribasés Marta, Ramos-Quiroga Josep Antoni, Buitelaar Jan K, Franke Barbara, Lesch Klaus-Peter

Abstract excerpt

BACKGROUND: Attention-deficit/hyperactivity disorder (ADHD) is a common, highly heritable neurodevelopmental disorder with profound cognitive, behavioral, and psychosocial impairments with persistence across the life cycle. Our initial genome-wide screening approach for copy number variants (CNVs) in ADHD implicated a duplication of SLC2A3, encoding glucose transporter-3 (GLUT3). GLUT3 plays a critical role in...

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PMID: 28224622
DOI: 10.1111/jcpp.12702

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

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