Filaggrin gene mutations and the distribution of filaggrin in oral mucosa of patients with oral lichen planus and healthy controls.

doi:10.1111/jdv.14098

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Community

Back to search

Article

Filaggrin gene mutations and the distribution of filaggrin in oral mucosa of patients with oral lichen planus and healthy controls.

Journal of the European Academy of Dermatology and Venereology : JEADV - 1 May 2017

Larsen K R, Johansen J D, Reibel J, Zachariae C, Rosing K, Pedersen A M L

Abstract excerpt

BACKGROUND: Lichen planus (LP) is a chronic inflammatory disease of unknown aetiology affecting the skin and oral mucosa. Oral lichenoid lesions (OLLs), like oral contact reactions, may resemble oral lichen planus (OLP) both clinically and histopathologically. As OLP and OLL are hyperkeratotic diseases and filaggrin is essential to keratinization, the distribution of filaggrin may be altered in these lesions....

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Aged
Share to Case-Control Studies
Share to Filaggrin Proteins

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to FLG gene

Share to Intermediate Filament Proteins

Share to Lichen Planus, Oral

Share to Mouth Mucosa

Share to Mutation

Source

Open in PubMed

Identifiers

PMID: 28000306
DOI: 10.1111/jdv.14098

Genes

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026.