Linc‐MAF‐4 regulates T h 1/T h 2 differentiation and is associated with the pathogenesis of multiple sclerosis by targeting MAF

Abstract excerpt

ABSTRACT In this study, we strove to substantiate the ability of linc‐MAF‐4 to act as a regulator of pathogenesis during multiple sclerosis (MS). We recruited 34 patients who were diagnosed with MS according to the revised McDonald criteria. Six patients with MS and 5 healthy volunteers contributed peripheral blood mononuclear cells for microarray analysis. Subsequent knockdown and overexpression of linc‐MAF‐4 in...

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