Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Mutations in ash1 and trx enhance P-element-dependent silencing in Drosophila melanogaster.

Genome - 1 Aug 2016

McCracken Allen, Locke John

Abstract excerpt

In Drosophila melanogaster, the mini-w(+) transgene in Pci is normally expressed throughout the adult eye; however, when other P or KP elements are present, a variegated-eye phenotype results, indicating random w(+) silencing during development called P-element-dependent silencing (PDS). Mutant Su(var)205 and Su(var)3-7 alleles act as haplo-suppressors/triplo-enhancers of this variegated phenotype, indicating...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Chromobox Protein Homolog 5
Share to Drosophila melanogaster
Share to Drosophila Proteins

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Gene Silencing

Share to Genetic Loci

Share to Heterochromatin

Share to Suppression, Genetic

Share to Transgenes

Source

Identifiers

PMID: 27373142
DOI: 10.1139/gen-2014-0127

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Mutations in ash1 and trx enhance P-element-dependent silencing in Drosophila melanogaster.PMID 27373142Read ↗

Mutations in CG8878, a novel putative protein kinase, enhance P element dependent silencing (PDS) and position effect variegation (PEV) in Drosophila melanogaster.Semantic proximity Read ↗

Comparative analysis of position-effect variegation mutations in Drosophila melanogaster delineates the targets of modifiers.Semantic proximity Read ↗

P-Element repression in Drosophila melanogaster by variegating clusters of P-lacZ-white transgenes.Semantic proximity Read ↗

P element-encoded regulatory products enhance Repeat-Induced Gene Silencing (RIGS) of P-lacZ-white clusters in Drosophila melanogaster.Semantic proximity Read ↗

pitkin(D), a novel gain-of-function enhancer of position-effect variegation, affects chromatin regulation during oogenesis and early embryogenesis in Drosophila.Semantic proximity Read ↗

Genetics of P-element transposition into Drosophila melanogaster centric heterochromatin.Semantic proximity Read ↗

Position-effect variegation, heterochromatin formation, and gene silencing in Drosophila.Semantic proximity Read ↗

Heterochromatin protein 1 modifies mammalian PEV in a dose- and chromosomal-context-dependent manner.Semantic proximity Read ↗

Genetic factors controlling white gene expression of the transposon A(R) 4-24 at a telomere in Drosophila melanogaster.Semantic proximity Read ↗

The impact of genetic background and cell lineage on the level and pattern of gene expression in position effect variegation.Semantic proximity Read ↗

A sex-influenced modifier in Drosophila that affects a broad spectrum of target loci including the histone repeats.Semantic proximity Read ↗

The Lighten up (Lip) gene of Drosophila melanogaster, a modifier of retroelement expression, position effect variegation and white locus insertion alleles.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.