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Article

Endothelial-to-mesenchymal transition in lipopolysaccharide-induced acute lung injury drives a progenitor cell-like phenotype.

American journal of physiology. Lung cellular and molecular physiology - 1 Jun 2016

Suzuki Toshio, Tada Yuji, Nishimura Rintaro, Kawasaki Takeshi, Sekine Ayumi, Urushibara Takashi, Kato Fumiaki, Kinoshita Taku, Ikari Jun, West James, Tatsumi Koichiro

Abstract excerpt

Pulmonary vascular endothelial function may be impaired by oxidative stress in endotoxemia-derived acute lung injury. Growing evidence suggests that endothelial-to-mesenchymal transition (EndMT) could play a pivotal role in various respiratory diseases; however, it remains unclear whether EndMT participates in the injury/repair process of septic acute lung injury. Here, we analyzed lipopolysaccharide...

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Identifiers

PMID: 27106288
DOI: 10.1152/ajplung.00074.2016

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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